CrossTalk proposal: Cell volume changes are an essential step in the cell death machinery.

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

Who cites it

12 citing papers in PubMed.

1. From Pinocytosis to Methuosis-Fluid Consumption as a Risk Factor for Cell Death.Frontiers in cell and developmental biology · 2021
   Review
2. Editorial: Ion and Water Transport in Cell Death.Frontiers in cell and developmental biology · 2021
   Article
3. Balance of NaFrontiers in cell and developmental biology · 2020
   Article
4. EphA4 receptor regulates outwardly rectifying chloride channel in CA1 hippocampal neurons after ischemia-reperfusion.Neuroreport · 2019
   Article
5. The Role of Chondrocyte Morphology and Volume in Controlling Phenotype-Implications for Osteoarthritis, Cartilage Repair, and Cartilage Engineering.Current rheumatology reports · 2019
   Review
6. Contribution of Anoctamins to Cell Survival and Cell Death.Cancers · 2019
   Review
7. Activation of a chondrocyte volume-sensitive Cl(-) conductance prior to macroscopic cartilage lesion formation in the rabbit knee anterior cruciate ligament transection osteoarthritis model.Osteoarthritis and cartilage · 2016
   Article
8. Cell volume control in three dimensions: Water movement without solute movement.The Journal of general physiology · 2015
   Article
9. An in vitro model of skeletal muscle volume regulation.PloS one · 2015
   Article
10. Syntaxin-binding domain of Kv2.1 is essential for the expression of apoptotic K+ currents.The Journal of physiology · 2014
    Article
11. The large conductance Ca(2+) -activated K(+) (BKCa) channel regulates cell proliferation in SH-SY5Y neuroblastoma cells by activating the staurosporine-sensitive protein kinases.Frontiers in physiology · 2014
    Article
12. Rebuttal from Sergei N. Orlov, Michael M. Model and Ryszard Grygorczyk.The Journal of physiology · 2013
    Article

Corrections and comments

• Commented on by
  Rebuttal from Sergei N. Orlov, Michael M. Model and Ryszard Grygorczyk.2013

Authors and funding

2 authors.

Funding

No grant is acknowledged in the PubMed record.

Abstract

PubMed holds no abstract for this paper.

Indexed as