ArticleProtoplasma2014
Programmed cell death during floral nectary senescence in Ipomoea purpurea.
Article in Protoplasma, 2014. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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Who cites it
6 citing papers in PubMed.
- Morphological Study on the Differentiation of Flower Buds and the Embryological Stages of Male and Female Floral Organs inPlants (Basel, Switzerland) · 2024Article
- Full-length RNA sequencing and single-nucleus sequencing deciphers programmed cell death and developmental trajectories in laticiferous canals ofFrontiers in plant science · 2024Article
- Developmental Programmed Cell Death Involved in Ontogenesis ofPlants (Basel, Switzerland) · 2023Article
- Functional Ecology of External Secretory Structures inPlants (Basel, Switzerland) · 2022Article
- The study of schizogenous formation of secretory ducts in Ferula ferulaeoides (Steud.) Korov.Protoplasma · 2022Article
- Tissue-specific expression of a soybean hypersensitive-induced response (HIR) protein gene promoter.Plant molecular biology · 2015Article
Corrections and comments
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Authors and funding
2 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
The nectaries of Ipomoea purpurea wilt in the late flowering period. The senescence process of nectaries is frequently associated with cell lysis. In this paper, various techniques were used to investigate whether programmed cell death (PCD) was involved in the senescence process of nectaries in I. purpurea. Ultrastructural studies showed that nectary cells began to undergo structural distortion, chromatin condensation, mitochondrial membrane degradation, and vacuolar-membrane dissolution and rupture after bloom. 4',6-Diamidino-2-phenylindole (DAPI) and terminal deoxynucleotidyl transferase-mediated 2'-deoxyuridine-5'-triphosphate (dUTP) nick end-labeling (TUNEL) assay showed that nectary cell nuclear DNA began to degrade during the budding stage, and disappeared in the fruiting stage. DNA gel electrophoresis showed that degradation of DNA was random. Together, these results suggest that PCD participate in the senescence of the nectary in I. purpurea. PCD began during the budding period, followed by significant changes in nectary morphology and structure during the flowering period. During the fruiting stage, the PCD process is complete and the nectary degrades.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.