Evidence map›Paper›PMID 24165757›Full record

ArticleThe pharmacogenomics journal2014

The association of cytochrome P450 genetic polymorphisms with sulfolane formation and the efficacy of a busulfan-based conditioning regimen in pediatric patients undergoing hematopoietic stem cell transplantation.

C R S Uppugunduri, M A Rezgui, P H Diaz, A K Tyagi, J Rousseau, Y Daali, M Duval, H Bittencourt, M Krajinovic, M Ansari

Registry-linked trialOpen access · bronzeAbstract read
PubMed Publisher
In one paragraph

Article in The pharmacogenomics journal, 2014. The graph could read no effect estimate from its abstract, so it casts no vote on the map. It is linked to trial NCT01257854 (Polymorphisms and Busulfan Pharmacokinetic Study in Pediatric Transplnatation), which is not on this map. Cited by 25 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
25citing papers in PubMed, 1 pooled it
2.3field-weighted citation impact, top 12% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

NCT01257854 unknown statusnot on this map

Polymorphisms and Busulfan Pharmacokinetic Study in Pediatric Transplnatation

TypeobservationalSponsorUniversity Hospital, GenevaRan2008 to 2023Enrolled200ConditionsChildren Who Receive a Stem Cell Transplantation With Busulfan IV
3 · Its place in the literature

Who cites it

25 citing papers in PubMed, 1 synthesis or guideline pooled it, 41 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

10 authors at 3 institutions in 3 countries.

C R S Uppugunduri1] Department of Pediatrics, Onco-Hematology unit, University Hospital of Geneva, Geneva, Switzerland [2] CANSEARCH Research Laboratory, Geneva Medical University, Geneva, Switzerland.
M A RezguiCharles-Bruneau Cancer Center, CHU Sainte-Justine Research Center, Montreal, Quebec, Canada.
P H Diaz1] Department of Pediatrics, Onco-Hematology unit, University Hospital of Geneva, Geneva, Switzerland [2] CANSEARCH Research Laboratory, Geneva Medical University, Geneva, Switzerland.
A K Tyagi1] Department of Pediatrics, Onco-Hematology unit, University Hospital of Geneva, Geneva, Switzerland [2] CANSEARCH Research Laboratory, Geneva Medical University, Geneva, Switzerland.
J RousseauCharles-Bruneau Cancer Center, CHU Sainte-Justine Research Center, Montreal, Quebec, Canada.
Y Daali1] CANSEARCH Research Laboratory, Geneva Medical University, Geneva, Switzerland [2] Department of Clinical Pharmacology and Toxicology, Geneva University Hospitals, Geneva, Switzerland.
M Duval1] Charles-Bruneau Cancer Center, CHU Sainte-Justine Research Center, Montreal, Quebec, Canada [2] Department of Pediatrics, Charles-Bruneau Cancer Center, CHU Sainte-Justine Research Center, Montreal, Quebec, Canada.
H Bittencourt1] Charles-Bruneau Cancer Center, CHU Sainte-Justine Research Center, Montreal, Quebec, Canada [2] Department of Pediatrics, Charles-Bruneau Cancer Center, CHU Sainte-Justine Research Center, Montreal, Quebec, Canada.
M Krajinovic1] Charles-Bruneau Cancer Center, CHU Sainte-Justine Research Center, Montreal, Quebec, Canada [2] Department of Pediatrics, Charles-Bruneau Cancer Center, CHU Sainte-Justine Research Center, Montreal, Quebec, Canada [3] Department of Pharmacology, University of Montreal, Montreal, Quebec, Canada.
M Ansari1] Department of Pediatrics, Onco-Hematology unit, University Hospital of Geneva, Geneva, Switzerland [2] CANSEARCH Research Laboratory, Geneva Medical University, Geneva, Switzerland.
Centre Hospitalier Universitaire Sainte-Justine · CAUniversity Hospital of Geneva · CHUniversity of Geneva · CH

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Cytochrome P450 enzymes (CYPs) and flavin-containing monooxygenases (FMOs) likely have a role in the oxidation of intermediate metabolites of busulfan (Bu). In vitro studies to investigate the involvement of these enzymes are cumbersome because of the volatile nature of the intermediate metabolite tetrahydrothiophene (THT) and the lack of sensitive quantitation methods. This study explored the association between the CYP2C9, CYP2C19, CYP2B6 and FMO3 genotypes and sulfolane (Su, a water soluble metabolite of Bu) plasma levels in children undergoing hematopoietic stem cell transplantation (HSCT). The relationship between these genotypes and the effectiveness of myeloablative conditioning was also analyzed. Sixty-six children receiving an intravenous Bu-based myeloablative conditioning regimen were genotyped for common functional variant alleles in CYP2C9 (*2 and *3), CYP2C19 (*2 and *17), FMO3 (rs2266780, rs2266782 and rs1736557) and CYP2B6 (*5 and *9). The plasma levels of Bu and its metabolite Su were measured after the ninth Bu dose in a subset of 44 patients for whom plasma samples were available. The ratio of Bu to Su was considered the metabolic ratio (MR) and was compared across the genotype groups. Higher MRs were observed in CYP2C9*2 and *3 allele carriers (mean±s.d.: 7.8±3.6 in carriers vs 4.4±2.2 in non-carriers; P=0.003). An increased incidence of graft failure was observed among patients with an MR>5 compared with those with MR values <5 (20% vs 0%; P=0.02). In contrast, a significantly higher incidence of relapse and graft failure (evaluated as event-free survival) was observed in patients with malignant disease who carried CYP2B6 alleles with reduced function on both chromosomes compared with carriers of at least one normal allele (100% vs 40%; P=0.0001). These results suggest that CYP2C9 has a role in the oxidation reactions of THT and indicate that it may be possible to predict the efficacy of Bu-based myeloablative conditioning before HSCT on the basis of CYP genotypes and Bu MRs.

Indexed as

Hematopoietic Stem Cell TransplantationPolymorphism, GeneticTransplantation ConditioningAdolescentAntineoplastic Agents, AlkylatingBusulfanChildChild, PreschoolCytochrome P-450 Enzyme SystemFemaleHumansInfantInfant, NewbornMaleThiophenesAntineoplastic Agents, AlkylatingBusulfanCytochrome P-450 Enzyme SystemsulfolaneThiophenes

Identifiers

PMID24165757
OpenAlexW2064320013

What OpenQuestion holds

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Registered trials

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.