Evidence map›Paper›PMID 23877837›Full record

ReviewInternational journal of molecular sciences2013

Chlamydia pneumoniae infection in atherosclerotic lesion development through oxidative stress: a brief overview.

Marisa Di Pietro, Simone Filardo, Fiorenzo De Santis, Rosa Sessa

Open access · goldAbstract readReview
In one paragraph

Review in International journal of molecular sciences, 2013. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 26 papers.

0numbers the graph read from it
0cells of the map it votes in
26citing papers in PubMed
5.7field-weighted citation impact, top 3% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

26 citing papers in PubMed, 47 citations in OpenAlex.

  1. Medicina (Kaunas, Lithuania) · 2026
    Article
  2. Article
  3. Frontiers in neuroscience · 2024
    Review
  4. Article
  5. Review
  6. Article
  7. Naringenin promotes cell autophagy to improve high-fat-diet-induced atherosclerosis in ApoE-/- mice.Brazilian journal of medical and biological research = Revista brasileira de pesquisas medicas e biologica · 2021
    Article
  8. Review
  9. Microorganisms · 2019
    Review
  10. Microorganisms · 2019
    Review
  11. Article
  12. Frontiers in aging neuroscience · 2018
    Review
  13. Review
  14. Article
  15. Review
  16. Article
  17. Article
  18. Article
  19. Article
  20. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

Marisa Di PietroDepartment of Public Health and Infectious Diseases, "Sapienza" University, Rome 00185, Italy.
Simone Filardo
Fiorenzo De Santis
Rosa Sessa

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Chlamydia pneumoniae, an obligate intracellular pathogen, is known as a leading cause of respiratory tract infections and, in the last two decades, has been widely associated with atherosclerosis by seroepidemiological studies, and direct detection of the microorganism within atheroma. C. pneumoniae is presumed to play a role in atherosclerosis for its ability to disseminate via peripheral blood mononuclear cells, to replicate and persist within vascular cells, and for its pro-inflammatory and angiogenic effects. Once inside the vascular tissue, C. pneumoniae infection has been shown to induce the production of reactive oxygen species in all the cells involved in atherosclerotic process such as macrophages, platelets, endothelial cells, and vascular smooth muscle cells, leading to oxidative stress. The aim of this review is to summarize the data linking C. pneumoniae-induced oxidative stress to atherosclerotic lesion development.

Indexed as

Oxidative StressAtherosclerosisChlamydia InfectionsChlamydophila pneumoniaeHumansMacrophagesMuscle, Smooth, VascularReactive Oxygen SpeciesReactive Oxygen Species

Identifiers

PMID23877837
PMCPMC3742290
OpenAlexW2069791273

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.