Evidence map›Paper›PMID 23770340›Full record

ArticleFree radical biology & medicine2013

Glutathione redox dynamics and expression of glutathione-related genes in the developing embryo.

Alicia R Timme-Laragy, Jared V Goldstone, Barry R Imhoff, John J Stegeman, Mark E Hahn, Jason M Hansen

Open access · greenAbstract read
In one paragraph

Article in Free radical biology & medicine, 2013. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 55 papers.

0numbers the graph read from it
0cells of the map it votes in
55citing papers in PubMed
5.9field-weighted citation impact, top 3% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

55 citing papers in PubMed, 132 citations in OpenAlex.

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  13. The Role of Oxidative Stress in Trisomy 21 Phenotype.Cellular and molecular neurobiology · 2023
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 3 institutions in 1 country.

Alicia R Timme-LaragyBiology Department, Woods Hole Oceanographic Institution, Woods Hole, MA 02543, USA; Division of Environmental Health, Department of Public Health, School of Public Health and Health Sciences, University of Massachusetts, Amherst, MA 01003, USA. Electronic address: aliciat@schoolph.umass.edu.
Jared V GoldstoneBiology Department, Woods Hole Oceanographic Institution, Woods Hole, MA 02543, USA.
Barry R ImhoffDivision of Pulmonary, Allergy/Immunology, Cystic Fibrosis and Sleep, Department of Pediatrics, Emory University, Atlanta, GA 30322, USA.
John J StegemanBiology Department, Woods Hole Oceanographic Institution, Woods Hole, MA 02543, USA.
Mark E HahnBiology Department, Woods Hole Oceanographic Institution, Woods Hole, MA 02543, USA.
Jason M HansenDivision of Pulmonary, Allergy/Immunology, Cystic Fibrosis and Sleep, Department of Pediatrics, Emory University, Atlanta, GA 30322, USA.
Woods Hole Oceanographic Institution · USEmory University · USUniversity of Massachusetts Amherst · US

Funding

Training CoreP42ES007381 · NIEHS · BOSTON UNIVERSITY MEDICAL CAMPUS · PI SHERR, DAVID H · 1995 to 2019
$46.1M
CYP Genes and the Generation of Oxidative StressR01ES015912 · NIEHS · WOODS HOLE OCEANOGRAPHIC INSTITUTION · PI STEGEMAN, JOHN J · 2007 to 2011
$3.1M
Mechanisms of Embryo Response to Oxidative StressR01ES016366 · NIEHS · WOODS HOLE OCEANOGRAPHIC INSTITUTION · PI HAHN, MARK E · 2009 to 2013
$1.9M
Differential Sensitivity to Oxidative Stress during Embryonic DevelopmentF32ES017585 · NIEHS · WOODS HOLE OCEANOGRAPHIC INSTITUTION · PI TIMME-LARAGY, ALICIA R · 2009 to 2011
$154k
NIEHS NIH HHS F32 ES017585NIEHS NIH HHS F32ES017585NIEHS NIH HHS P42 ES007381NIEHS NIH HHS R01 ES015912NIEHS NIH HHS R01ES015912NIEHS NIH HHS R01 ES016366NIEHS NIH HHS R01ES016366
6 · The paper itself

Abstract

Embryonic development involves dramatic changes in cell proliferation and differentiation that must be highly coordinated and tightly regulated. Cellular redox balance is critical for cell fate decisions, but it is susceptible to disruption by endogenous and exogenous sources of oxidative stress. The most abundant endogenous nonprotein antioxidant defense molecule is the tripeptide glutathione (γ-glutamylcysteinylglycine, GSH), but the ontogeny of GSH concentration and redox state during early life stages is poorly understood. Here, we describe the GSH redox dynamics during embryonic and early larval development (0-5 days postfertilization) in the zebrafish (Danio rerio), a model vertebrate embryo. We measured reduced and oxidized glutathione using HPLC and calculated the whole embryo total glutathione (GSHT) concentrations and redox potentials (Eh) over 0-120 h of zebrafish development (including mature oocytes, fertilization, midblastula transition, gastrulation, somitogenesis, pharyngula, prehatch embryos, and hatched eleutheroembryos). GSHT concentration doubled between 12h postfertilization (hpf) and hatching. The GSH Eh increased, becoming more oxidizing during the first 12h, and then oscillated around -190 mV through organogenesis, followed by a rapid change, associated with hatching, to a more negative (more reducing) Eh (-220 mV). After hatching, Eh stabilized and remained steady through 120 hpf. The dynamic changes in GSH redox status and concentration defined discrete windows of development: primary organogenesis, organ differentiation, and larval growth. We identified the set of zebrafish genes involved in the synthesis, utilization, and recycling of GSH, including several novel paralogs, and measured how expression of these genes changes during development. Ontogenic changes in the expression of GSH-related genes support the hypothesis that GSH redox state is tightly regulated early in development. This study provides a foundation for understanding the redox regulation of developmental signaling and investigating the effects of oxidative stress during embryogenesis.

Indexed as

Gene Expression Regulation, DevelopmentalAnimalsEmbryo, NonmammalianGlutathioneOligonucleotide Array Sequence AnalysisOxidation-ReductionZebrafishGlutathioneAntioxidantEmbryonic developmentFree radicalsGclGclcGclmGene expressionglutamate–cysteine ligaseglutamate–cysteine ligase catalytic subunitglutamate–cysteine ligase modifier subunitGlutathioneglutathione disulfideglutathione synthetaseGSHGssGSSGOxidative stressPCAperchloric acidreactive oxygen speciesRedoxreduced glutathioneROSZebrafish

Identifiers

PMID23770340
PMCPMC3823629
OpenAlexW2095466273

What OpenQuestion holds

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Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.