Evidence map›Paper›PMID 23690608›Full record

ArticleProceedings of the National Academy of Sciences of the United States of America2013

Genetic screens to identify pathogenic gene variants in the common cancer predisposition Lynch syndrome.

Mark Drost, Anne Lützen, Sandrine van Hees, Daniel Ferreira, Fabienne Calléja, José B M Zonneveld, Finn Cilius Nielsen, Lene Juel Rasmussen, Niels de Wind

Abstract read
In one paragraph

Article in Proceedings of the National Academy of Sciences of the United States of America, 2013. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 11 papers.

0numbers the graph read from it
0cells of the map it votes in
11citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

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Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

11 citing papers in PubMed.

  1. Article
  2. Article
  3. A RareGenes · 2022
    Article
  4. Article
  5. Article
  6. Coordinated roles of SLX4 and MutSβ in DNA repair and the maintenance of genome stability.Critical reviews in biochemistry and molecular biology · 2021
    Review
  7. Article
  8. Article
  9. Review
  10. Article
  11. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

9 authors.

Mark DrostDepartment of Toxicogenetics, Leiden University Medical Center, 2300 RC, Leiden, The Netherlands.
Anne Lützen
Sandrine van Hees
Daniel Ferreira
Fabienne Calléja
José B M Zonneveld
Finn Cilius Nielsen
Lene Juel Rasmussen
Niels de Wind

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

In many individuals suspected of the common cancer predisposition Lynch syndrome, variants of unclear significance (VUS), rather than an obviously pathogenic mutations, are identified in one of the DNA mismatch repair (MMR) genes. The uncertainty of whether such VUS inactivate MMR, and therefore are pathogenic, precludes targeted healthcare for both carriers and their relatives. To facilitate the identification of pathogenic VUS, we have developed an in cellulo genetic screen-based procedure for the large-scale mutagenization, identification, and cataloging of residues of MMR genes critical for MMR gene function. When a residue identified as mutated in an individual suspected of Lynch syndrome is listed as critical in such a reverse diagnosis catalog, there is a high probability that the corresponding human VUS is pathogenic. To investigate the applicability of this approach, we have generated and validated a prototypic reverse diagnosis catalog for the MMR gene MutS Homolog 2 (Msh2) by mutagenizing, identifying, and cataloging 26 deleterious mutations in 23 amino acids. Extensive in vivo and in vitro analysis of mutants listed in the catalog revealed both recessive and dominant-negative phenotypes. Nearly half of these critical residues match with VUS previously identified in individuals suspected of Lynch syndrome. This aids in the assignment of pathogenicity to these human VUS and validates the approach described here as a diagnostic tool. In a wider perspective, this work provides a model for the translation of personalized genomics into targeted healthcare.

Indexed as

Genetic VariationModels, MolecularAmino Acid SubstitutionBase SequenceColorectal Neoplasms, Hereditary NonpolyposisGenetic Predisposition to DiseaseGenetic TestingHumansMicrosatellite RepeatsMolecular Sequence DataMutagenesisMutationMutS Homolog 2 ProteinReverse GeneticsSequence Analysis, DNAThioguanineMSH2 protein, humanMutS Homolog 2 ProteinThioguanine

Identifiers

PMID23690608
PMCPMC3677471

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.