Evidence map›Paper›PMID 23635943›Full record

ReviewJournal of innate immunity2013

Control of myeloid cell trafficking in resolution.

Lucy V Norling, Mauro Perretti

Open access · bronzeAbstract readReview
In one paragraph

Review in Journal of innate immunity, 2013. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 16 papers.

0numbers the graph read from it
0cells of the map it votes in
16citing papers in PubMed
2.5field-weighted citation impact, top 11% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

16 citing papers in PubMed, 27 citations in OpenAlex.

  1. Article
  2. Article
  3. Article
  4. Article
  5. Review
  6. Review
  7. Review
  8. Article
  9. Article
  10. Nonredundant protective properties of FPR2/ALX in polymicrobial murine sepsis.Proceedings of the National Academy of Sciences of the United States of America · 2014
    Article
  11. Article
  12. Article
  13. Review
  14. Article
  15. Article
  16. Myeloid cells in traffic.Journal of innate immunity · 2013
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors at 2 institutions in 2 countries.

Lucy V NorlingThe William Harvey Research Institute, Barts and The London School of Medicine and Dentistry, London, UK. l.v.norling@qmul.ac.uk
Mauro Perretti
Karger Publishers (Switzerland) · CHWilliam Harvey Research Institute · GB

Funding

Arthritis Research UK 19909Medical Research Council MR/K013068/1Versus Arthritis 19909Wellcome Trust 086867/Z/08/Z
6 · The paper itself

Abstract

Following tissue injury or microbial invasion, neutrophils are robustly recruited to inflammatory loci, which is a hallmark of the host inflammatory response. This event initiates a series of processes required to activate resolution, including recruitment of monocytes, clearance of microbes, cellular debris and apoptotic neutrophils, the egress of phagocytes and, ultimately, regain of tissue homeostasis. Substantial evidence now signifies that resolution of inflammation is a highly coordinated, active process dictated by the spatial-temporal generation of proresolving mediators that act on specific receptors to modulate cell and tissue reactivity. This review will focus on the mediators, targets and pathways initiated to orchestrate resolution. Importantly, disruption of the key processes involved in inflammatory resolution could result in delayed restoration of tissue homeostasis, leading to fibrosis and/or persistent inflammation.

Indexed as

AnimalsCell Adhesion MoleculesCell CommunicationCell MovementHomeostasisHumansInflammationMonocytesMyeloid CellsPhagocytosisReceptor Cross-TalkSignal TransductionCell Adhesion Molecules

Identifiers

PMID23635943
PMCPMC6741450
OpenAlexW2078653493

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.