ArticleFrontiers in plant science2013
The xylem as battleground for plant hosts and vascular wilt pathogens.
Article in Frontiers in plant science, 2013. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 203 papers, 1 of them a synthesis that pooled it.
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Who cites it
203 citing papers in PubMed, 1 synthesis or guideline pooled it.
- A systematic review of multi-mode analytics for enhanced plant stress evaluation.Frontiers in plant science · 2025Pooled it
- The complex subunit VdAP-2α regulates polar growth, ergosterol metabolism, and virulence inVirulence · 2026Article
- Invasion of Clavibacter michiganensis into the Xylem and Surrounding Tissues Drives Histological Alterations and Disease Development in Tomato.The plant pathology journal · 2026Article
- Hijacked hydraulics: Verticillium dahliae-induced xylem dysfunction in pepper stems revealed by integrated hydraulic, imaging, and molecular analyses.The New phytologist · 2026Article
- The Conserved Guanyl-Specific Ribonuclease T1 Effector EC19 Is Required for Full Virulence of the Vascular Wilt Fungus Fusarium oxysporum on Tomato.Molecular plant pathology · 2026Article
- Attention-Enhanced YOLOv11 for Early Detection of Fungal-Induced Forest Tree Decline.Plants (Basel, Switzerland) · 2026Article
- Beneath the Bark: Molecular Determinants of Adaptation and Infection in Bacterial Pathogens of Woody Hosts.Molecular plant pathology · 2026Review
- Exploring the biochemical and biological functions of copper radical oxidases in the vascular wilt phytopathogenApplied and environmental microbiology · 2026Article
- Article
- Effects of Musts from Esca-Proper-Affected 'Primitivo' Vines on Two CommercialPlants (Basel, Switzerland) · 2026Article
- Comparative genomics of strains of varying virulence of the gram-positive bacterial pathogen of maize, Clavibacter nebraskensis.BMC genomics · 2026Article
- Computational identification of inhibitors from Aegle marmelos leaf extract targeting ergosterol biosynthesis ERG4/ERG24 enzyme and serine proteases for combatting mancozeb-resistant Fusarium.Antonie van Leeuwenhoek · 2026Article
- Shaping future forests: how can ecophysiology support climate-smart forest management?The New phytologist · 2026Review
- Systemic Delivery of Functional Proteins Into Plants Using an Engineered Membrane Translocation Domain.Plant biotechnology journal · 2026Article
- Phase-contrast microtomography unveils mechanisms of root colonization by a vascular fungal pathogen.Nature communications · 2026Article
- Mixed-Pathogen Infections in Vegetatively Propagated Crops: From Biological Synergism to Integrated Management.Plants (Basel, Switzerland) · 2026Review
- Lecanicillium aphanocladii-derived copper oxide nanoparticles as a green nano-fungicide enhancing molecular and antioxidant defense against fusarium wilt in potato.Microbial cell factories · 2026Article
- Physiological and Metabolic Mechanisms ofJournal of fungi (Basel, Switzerland) · 2026Article
- Article
- Tissue-specific experimental evolution reveals adaptive trade-offs in the plant vascular pathogen Clavibacter michiganensis.The ISME journal · 2026Article
143 more citing papers are in PubMed but not listed here.
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Authors and funding
2 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Vascular wilts are among the most destructive plant diseases that occur in annual crops as well as in woody perennials. These diseases are generally caused by soil-borne bacteria, fungi, and oomycetes that infect through the roots and enter the water-conducting xylem vessels where they proliferate and obstruct the transportation of water and minerals. As a consequence, leaves wilt and die, which may lead to impairment of the whole plant and eventually to death of the plant. Cultural, chemical, and biological measures to control this group of plant pathogens are generally ineffective, and the most effective control strategy is the use of genetic resistance. Owing to the fact that vascular wilt pathogens live deep in the interior of their host plants, studies into the biology of vascular pathogens are complicated. However, to design novel strategies to combat vascular wilt diseases, understanding the (molecular) biology of vascular pathogens and the molecular mechanisms underlying plant defense against these pathogens is crucial. In this review, we discuss the current knowledge on interactions of vascular wilt pathogens with their host plants, with emphasis on host defense responses against this group of pathogens.
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