ArticleFrontiers in computational neuroscience2013
Cortical information flow in Parkinson's disease: a composite network/field model.
Article in Frontiers in computational neuroscience, 2013. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 18 papers.
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Who cites it
18 citing papers in PubMed.
- Dysfunction of motor cortices in Parkinson's disease.Cerebral cortex (New York, N.Y. : 1991) · 2024Review
- Dynamics of parkinsonian oscillations mediated by transmission delays in a mean-field model of the basal ganglia.Frontiers in cellular neuroscience · 2024Article
- Ultrafast simulation of large-scale neocortical microcircuitry with biophysically realistic neurons.eLife · 2022Article
- Neuro4PD: An Initial Neurorobotics Model of Parkinson's Disease.Frontiers in neurorobotics · 2021Article
- Dysregulation of excitatory neural firing replicates physiological and functional changes in aging visual cortex.PLoS computational biology · 2021Article
- NFTsim: Theory and Simulation of Multiscale Neural Field Dynamics.PLoS computational biology · 2018Article
- Article
- Optimization by Adaptive Stochastic Descent.PloS one · 2018Article
- Credibility, Replicability, and Reproducibility in Simulation for Biomedicine and Clinical Applications in Neuroscience.Frontiers in neuroinformatics · 2018Review
- The role of cortical oscillations in a spiking neural network model of the basal ganglia.PloS one · 2017Article
- Multitarget Multiscale Simulation for Pharmacological Treatment of Dystonia in Motor Cortex.Frontiers in pharmacology · 2016Article
- Computer modeling for pharmacological treatments for dystonia.Drug discovery today. Disease models · 2016Article
- Functional correlates of exaggerated oscillatory activity in basal ganglia output in hemiparkinsonian rats.Experimental neurology · 2014Article
- Towards real-time communication betweenJournal of computational surgery · 2014Article
- Neural masses and fields: modeling the dynamics of brain activity.Frontiers in computational neuroscience · 2014Article
- Reinforcement learning of two-joint virtual arm reaching in a computer model of sensorimotor cortex.Neural computation · 2013Article
- Reaction-diffusion in the NEURON simulator.Frontiers in neuroinformatics · 2013Article
- Multiscale modeling for clinical translation in neuropsychiatric disease.Journal of computational surgeryArticle
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6 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
The basal ganglia play a crucial role in the execution of movements, as demonstrated by the severe motor deficits that accompany Parkinson's disease (PD). Since motor commands originate in the cortex, an important question is how the basal ganglia influence cortical information flow, and how this influence becomes pathological in PD. To explore this, we developed a composite neuronal network/neural field model. The network model consisted of 4950 spiking neurons, divided into 15 excitatory and inhibitory cell populations in the thalamus and cortex. The field model consisted of the cortex, thalamus, striatum, subthalamic nucleus, and globus pallidus. Both models have been separately validated in previous work. Three field models were used: one with basal ganglia parameters based on data from healthy individuals, one based on data from individuals with PD, and one purely thalamocortical model. Spikes generated by these field models were then used to drive the network model. Compared to the network driven by the healthy model, the PD-driven network had lower firing rates, a shift in spectral power toward lower frequencies, and higher probability of bursting; each of these findings is consistent with empirical data on PD. In the healthy model, we found strong Granger causality between cortical layers in the beta and low gamma frequency bands, but this causality was largely absent in the PD model. In particular, the reduction in Granger causality from the main "input" layer of the cortex (layer 4) to the main "output" layer (layer 5) was pronounced. This may account for symptoms of PD that seem to reflect deficits in information flow, such as bradykinesia. In general, these results demonstrate that the brain's large-scale oscillatory environment, represented here by the field model, strongly influences the information processing that occurs within its subnetworks. Hence, it may be preferable to drive spiking network models with physiologically realistic inputs rather than pure white noise.
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