Evidence map›Paper›PMID 23604588›Full record

ArticleActa neuropathologica2013

Anle138b: a novel oligomer modulator for disease-modifying therapy of neurodegenerative diseases such as prion and Parkinson's disease.

Jens Wagner, Sergey Ryazanov, Andrei Leonov, Johannes Levin, Song Shi, Felix Schmidt, Catharina Prix, Francisco Pan-Montojo, Uwe Bertsch, Gerda Mitteregger-Kretzschmar and 22 more

3 registry-linked trialsOpen access · hybridAbstract read
In one paragraph

Article in Acta neuropathologica, 2013. The graph could read no effect estimate from its abstract, so it casts no vote on the map. It is linked to 3 registered trials, which are not on this map. Cited by 244 papers, 2 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
244citing papers in PubMed, 2 pooled it
10.6field-weighted citation impact, top 1% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

NCT04208152 phase1completednot on this mapstarted 2019, after this paper: background citation

A First-in-Human Study to Assess the Safety, Tolerability and Pharmacokinetics of anle138b in Healthy Male and Female Subjects

TypeinterventionalSponsorMODAG GmbHRan2019 to 2020Enrolled68ConditionsHealthy VolunteersArmsanle138b, Placebo
NCT04685265 phase1completednot on this mapstarted 2020, after this paper: background citation

A Study to Assess the Safety, Tolerability, Pharmacokinetics and Pharmacodynamics of Multiple Ascending Oral Doses of anle138b, and to Characterise the Effect of Food of anle138b in Mild to Moderate Parkinson's Disease

TypeinterventionalSponsorMODAG GmbHRan2020 to 2022Enrolled70ConditionsParkinson DiseaseArmsanle138b, Placebo
NCT05532358 phase1completednot on this mapstarted 2022, after this paper: background citation

An Open-Label, One-Sequence, Two-Part Drug-Drug Interaction Study in Healthy Volunteers to Assess the CYP1A2 and CYP3A4 Perpetrator Interaction Potential and CYP1A2 Victim Potential of TEV-56286 (anle138b)

TypeinterventionalSponsorMODAG GmbHRan2022 to 2023Enrolled54ConditionsHealthy VolunteersArmsanle138b (TEV-56286), Fluvoxamine 100 mg QD for 5 days
3 · Its place in the literature

Who cites it

244 citing papers in PubMed, 2 syntheses or guidelines pooled it, 447 citations in OpenAlex.

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184 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

32 authors at 10 institutions in 1 country.

Jens WagnerZentrum für Neuropathologie und Prionforschung, Ludwig-Maximilians-Universität München, Feodor-Lynen-Str. 23, 81377, Munich, Germany.
Sergey Ryazanov
Andrei Leonov
Johannes Levin
Song Shi
Felix Schmidt
Catharina Prix
Francisco Pan-Montojo
Uwe Bertsch
Gerda Mitteregger-Kretzschmar
Markus Geissen
Martin Eiden
Fabienne Leidel
Thomas Hirschberger
Andreas A Deeg
Julian J Krauth
Wolfgang Zinth
Paul Tavan
Jens Pilger
Markus Zweckstetter
Tobias Frank
Mathias Bähr
Jochen H Weishaupt
Manfred Uhr
Henning Urlaub
Ulrike Teichmann
Matthias Samwer
Kai Bötzel
Martin Groschup
Hans Kretzschmar
Christian Griesinger
Armin Giese
Ludwig-Maximilians-Universität München · DEMax Planck Institute for Biophysical Chemistry · DEFriedrich-Loeffler-Institut · DENanoscale Microscopy and Molecular Physiology of the Brain Cluster of Excellence 171 — DFG Research Center 103 · DEUniversitätsmedizin Göttingen · DELMU Klinikum · DEMax Planck Institute of Psychiatry · DETU Dresden · DEUniversity Hospital Schleswig-Holstein · DEUniversity of Göttingen · DE

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

In neurodegenerative diseases such as Alzheimer's disease (AD), Parkinson's disease (PD) and prion diseases, deposits of aggregated disease-specific proteins are found. Oligomeric aggregates are presumed to be the key neurotoxic agent. Here we describe the novel oligomer modulator anle138b [3-(1,3-benzodioxol-5-yl)-5-(3-bromophenyl)-1H-pyrazole], an aggregation inhibitor we developed based on a systematic high-throughput screening campaign combined with medicinal chemistry optimization. In vitro, anle138b blocked the formation of pathological aggregates of prion protein (PrP(Sc)) and of α-synuclein (α-syn), which is deposited in PD and other synucleinopathies such as dementia with Lewy bodies (DLB) and multiple system atrophy (MSA). Notably, anle138b strongly inhibited all prion strains tested including BSE-derived and human prions. Anle138b showed structure-dependent binding to pathological aggregates and strongly inhibited formation of pathological oligomers in vitro and in vivo both for prion protein and α-synuclein. Both in mouse models of prion disease and in three different PD mouse models, anle138b strongly inhibited oligomer accumulation, neuronal degeneration, and disease progression in vivo. Anle138b had no detectable toxicity at therapeutic doses and an excellent oral bioavailability and blood-brain-barrier penetration. Our findings indicate that oligomer modulators provide a new approach for disease-modifying therapy in these diseases, for which only symptomatic treatment is available so far. Moreover, our findings suggest that pathological oligomers in neurodegenerative diseases share structural features, although the main protein component is disease-specific, indicating that compounds such as anle138b that modulate oligomer formation by targeting structure-dependent epitopes can have a broad spectrum of activity in the treatment of different protein aggregation diseases.

Indexed as

alpha-SynucleinAnimalsBrainCell Culture TechniquesDisease Models, AnimalFemaleHumansMiceMice, Inbred C57BLParkinson DiseasePrion DiseasesPrionsPyrazolesPyrimidinesRotenonealpha-SynucleinN(4)-benzyl-N(6),N(6)-dimethyl-1-1(tert-butyl)-1H-pyrazolo(3,4-d)pyrimidine-6,4-diaminePrionsPyrazolesPyrimidinesRotenone

Identifiers

PMID23604588
PMCPMC3661926
OpenAlexW2034075546

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.