Evidence map›Paper›PMID 23582962›Full record

ReviewSeminars in perinatology2013

Progress in understanding the genetics of bronchopulmonary dysplasia.

Gary M Shaw, Hugh M O'Brodovich

Open access · greenAbstract readReview
In one paragraph

Review in Seminars in perinatology, 2013. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 20 papers.

0numbers the graph read from it
0cells of the map it votes in
20citing papers in PubMed
6.9field-weighted citation impact, top 3% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

20 citing papers in PubMed, 62 citations in OpenAlex.

  1. Article
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  6. Article
  7. Review
  8. Can We Prevent Bronchopulmonary Dysplasia?The Journal of pediatrics · 2017
    Review
  9. Article
  10. Review
  11. Article
  12. Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors at 1 institution in 1 country.

Gary M ShawDepartment of Pediatrics, Stanford University School of Medicine, Palo Alto, CA 94305, USA.
Hugh M O'Brodovich
Stanford University · US

Funding

Genomic Influences on Bronchopulmonary Dysplasia-A Population-Based StudyRC2HL101748 · NHLBI · STANFORD UNIVERSITY · PI OBRODOVICH, HUGH · 2009 to 2010
$3.5M
NHLBI NIH HHS RC2 HL101748NHLBI NIH HHS RC2HL101748
6 · The paper itself

Abstract

Bronchopulmonary dysplasia (BPD) is the most common chronic lung disease in infants. Its treatment imposes considerable healthcare burden and costs in the perinatal and early childhood period and patients are usually left with lifelong deficits in lung function. Evidence exists for different pathophysiologic pathways that can promote the structural changes that characterize BPD, including the impairment in alveolarization; however, there is increasing interest regarding heritable factors that may predispose very low birth weight infants to BPD. Our review focuses on recent publications that have investigated genetic factors that may potentially contribute to such reported heritability. These publications point us toward some possible genomic candidates for further study, but certainly do not identify any particular gene or gene pathway that would be inferred to be contributing substantially to the underlying etiology of BPD.

Indexed as

Bronchopulmonary DysplasiaGenetic Predisposition to DiseaseGenome-Wide Association StudyHumansInfant, NewbornInfant, PrematureInfant, Very Low Birth WeightPolymorphism, Single Nucleotide

Identifiers

PMID23582962
PMCPMC3628629
OpenAlexW1964538319

What OpenQuestion holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.