ArticleJournal of molecular modeling2013
Mechanisms on inhibition of polyethylene electrical tree aging: a theoretical study.
Article in Journal of molecular modeling, 2013. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
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Who cites it
1 citing paper in PubMed.
- Theoretical study on the mechanisms of polyethylene electrical breakdown strength increment by the addition of voltage stabilizers.Journal of molecular modeling · 2014Article
Corrections and comments
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Authors and funding
5 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
A theoretical investigation is completed on inhibition mechanism of polyethylene electrical tree aging. Foremost it elucidates that it is one of the important factors for inhibiting initiation and propagation of polyethylene electrical tree through keto-enol tautomerism of acetophenone and its analogues. Geometries of the keto tautomer and the enol tautomer of acetophenone and its analogues, and its transition states are optimized at the B3LYP/6-311+G(d,p) level, the harmonic vibration frequencies of the equilibrium geometries are calculated at the same level. The minimum energy path (MEP) is obtained by the intrinsic reaction coordinate (IRC) theory at the same level. The calculated results show that the energy barriers of keto-enol tautomerism of acetophenone and its analogues are smaller than the average C-C bond energy of polyethylene, the acetophenone and its analogues adulterated in polyethylene composites can improve the strength of alternate current puncture that PE can endure as well as inhibit polyethylene electrical tree from initiation and propagation.
Identifiers
23579420What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.