Evidence map›Paper›PMID 23546636›Full record

ArticleJournal of mathematical biology2014

Multiscale analysis for a vector-borne epidemic model.

Max O Souza

Abstract read
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In one paragraph

Article in Journal of mathematical biology, 2014. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.

0numbers the graph read from it
0cells of the map it votes in
3citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

3 citing papers in PubMed.

  1. Discrete epidemic models with two time scales.Advances in difference equations · 2021
    Article
  2. State estimators for some epidemiological systems.Journal of mathematical biology · 2019
    Article
  3. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

1 author.

Max O SouzaDepartamento de Matemática Aplicada, Universidade Federal Fluminense, R. Mário Santos Braga, s/n, Niterói, RJ, 22240-920, Brazil, msouza@mat.uff.br.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Traditional studies about disease dynamics have focused on global stability issues, due to their epidemiological importance. We study a classical SIR-SI model for arboviruses in two different directions: we begin by describing an alternative proof of previously known global stability results by using only a Lyapunov approach. In the sequel, we take a different view and we argue that vectors and hosts can have very distinctive intrinsic time-scales, and that such distinctiveness extends to the disease dynamics. Under these hypothesis, we show that two asymptotic regimes naturally appear: the fast host dynamics and the fast vector dynamics. The former regime yields, at leading order, a SIR model for the hosts, but with a rational incidence rate. In this case, the vector disappears from the model, and the dynamics is similar to a directly contagious disease. The latter yields a SI model for the vectors, with the hosts disappearing from the model. Numerical results show the performance of the approximation, and a rigorous proof validates the reduced models.

Indexed as

EpidemicsModels, BiologicalAnimalsArbovirusesArbovirus InfectionsArthropod VectorsComputer SimulationHost-Pathogen InteractionsHumans

Identifiers

What OpenQuestion holds

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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.