Evidence map›Paper›PMID 23440217›Full record

ArticleProceedings of the National Academy of Sciences of the United States of America2013

Direct involvement of DprA, the transformation-dedicated RecA loader, in the shut-off of pneumococcal competence.

Nicolas Mirouze, Mathieu A Bergé, Anne-Lise Soulet, Isabelle Mortier-Barrière, Yves Quentin, Gwennaele Fichant, Chantal Granadel, Marie-Françoise Noirot-Gros, Philippe Noirot, Patrice Polard and 2 more

Abstract read
In one paragraph

Article in Proceedings of the National Academy of Sciences of the United States of America, 2013. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 64 papers.

0numbers the graph read from it
0cells of the map it votes in
64citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

64 citing papers in PubMed.

  1. Review
  2. Article
  3. Characterisation of the NovelInternational journal of molecular sciences · 2025
    Article
  4. Article
  5. Article
  6. Article
  7. DprA recruits ComM to facilitate recombination during natural transformation in Gram-negative bacteria.Proceedings of the National Academy of Sciences of the United States of America · 2025
    Article
  8. Comparative analysis of theFrontiers in microbiology · 2025
    Article
  9. Competence Regulation inCurrent medicinal chemistry · 2025
    Review
  10. Article
  11. Article
  12. Article
  13. Investigating theMicrobiology (Reading, England) · 2022
    Article
  14. Article
  15. Article
  16. Attenuating theJournal of medicinal chemistry · 2022
    Article
  17. Article
  18. The transcription regulator BrsR serves as a network hub of natural competence protein-protein interactions inProceedings of the National Academy of Sciences of the United States of America · 2021
    Article
  19. Article
  20. Strategies to Attenuate the Competence Regulon inPeptide science (Hoboken, N.J.) · 2021
    Article

4 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

12 authors.

Nicolas MirouzeLaboratoire de Microbiologie et Génétique Moléculaires-Unité Mixte de Recherche 5100, Centre National de la Recherche Scientifique, F-31000 Toulouse, France.
Mathieu A Bergé
Anne-Lise Soulet
Isabelle Mortier-Barrière
Yves Quentin
Gwennaele Fichant
Chantal Granadel
Marie-Françoise Noirot-Gros
Philippe Noirot
Patrice Polard
Bernard Martin
Jean-Pierre Claverys

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Natural bacterial transformation is a genetically programmed process allowing genotype alterations that involves the internalization of DNA and its chromosomal integration catalyzed by the universal recombinase RecA, assisted by its transformation-dedicated loader, DNA processing protein A (DprA). In Streptococcus pneumoniae, the ability to internalize DNA, known as competence, is transient, developing suddenly and stopping as quickly. Competence is induced by the comC-encoded peptide, competence stimulating peptide (CSP), via a classic two-component regulatory system ComDE. Upon CSP binding, ComD phosphorylates the ComE response-regulator, which then activates transcription of comCDE and the competence-specific σ(X), leading to a sudden rise in CSP levels and rendering all cells in a culture competent. However, how competence stops has remained unknown. We report that DprA, under σ(X) control, interacts with ComE∼P to block ComE-driven transcription, chiefly impacting σ(X) production. Mutations of dprA specifically disrupting interaction with ComE were isolated and shown to map mainly to the N-terminal domain of DprA. Wild-type DprA but not ComE interaction mutants affected in vitro binding of ComE to its promoter targets. Once introduced at the dprA chromosomal locus, mutations disrupting DprA interaction with ComE altered competence shut-off. The absence of DprA was found to negatively impact growth following competence induction, highlighting the importance of DprA for pneumococcal physiology. DprA has thus two key roles: ensuring production of transformants via interaction with RecA and competence shut-off via interaction with ComE, avoiding physiologically detrimental consequences of prolonged competence. Finally, phylogenetic analyses revealed that the acquisition of a new function by DprA impacted its evolution in streptococci relying on ComE to regulate comX expression.

Indexed as

Bacterial ProteinsDNA Transformation CompetenceGene Expression Regulation, BacterialMembrane ProteinsMutationProtein Structure, TertiaryRec A RecombinasesStreptococcus pneumoniaeTranscription FactorsTranscription, GeneticBacterial ProteinsComE protein, Streptococcus pneumoniaecompetence factor, StreptococcusComX protein, StreptococcusDprA protein, bacteriaMembrane ProteinsRec A RecombinasesTranscription Factors

Identifiers

PMID23440217
PMCPMC3600483

What OpenQuestion holds

Textmetadata
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Registered trials

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Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.