Mitochondria in hypoxic pulmonary vasoconstriction: potential importance of compartmentalized reactive oxygen species signaling.

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

Who cites it

5 citing papers in PubMed.

1. Contribution of Mitochondrial Reactive Oxygen Species to Chronic Hypoxia-Induced Pulmonary Hypertension.Antioxidants (Basel, Switzerland) · 2023
   Article
2. Mitochondrial Dysfunction in Pulmonary Hypertension.Antioxidants (Basel, Switzerland) · 2023
   Review
3. Reactive oxygen species effect PASMCs apoptosis via regulation of dynamin-related protein 1 in hypoxic pulmonary hypertension.Histochemistry and cell biology · 2016
   Article
4. Targeting mitochondrial reactive oxygen species to modulate hypoxia-induced pulmonary hypertension.Free radical biology & medicine · 2015
   Article
5. Update in pulmonary vascular diseases 2013.American journal of respiratory and critical care medicine · 2014
   Review

Corrections and comments

• Comment on
  Superoxide generated at mitochondrial complex III triggers acute responses to hypoxia in the pulmonary circulation.2013

Authors and funding

3 authors.

Funding

Abstract

PubMed holds no abstract for this paper.

Indexed as