ArticleBMC nephrology2013
Autosomal dominant polycystic kidney disease with ectopic unilateral multicystic dysplastic kidney.
Article in BMC nephrology, 2013. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
6 citing papers in PubMed, 6 citations in OpenAlex.
- Prenatal Diagnosis and Postnatal Outcomes of Fetal ADPKD: A Single-Center Retrospective Cohort Study.Medicina (Kaunas, Lithuania) · 2025Article
- Autosomal dominant polycystic kidney disease with ectopic unilateral multicystic kidney: a case report.Journal of medical case reports · 2024Article
- Pancreatic extracorporeal shock wave lithotripsy for a patient concurrent with autosomal dominant polycystic kidney disease: a case report.The Journal of international medical research · 2022Article
- A case of unilateral nephrectomy performed for autosomal dominant polycystic kidney disease with marked unilateral enlargement.CEN case reports · 2018Article
- Minimally invasive open nephrectomy on children with multicystic dysplastic kidney.Experimental and therapeutic medicine · 2016Article
- Long-term follow up of a boy with unilateral autosomal dominant polycystic kidney disease and contralateral renal agenesis.Developmental period medicineArticle
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Authors and funding
8 authors at 3 institutions in 1 country.
Funding
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Abstract
backgroundAutosomal dominant polycystic kidney disease (ADPKD) is the most common hereditary renal disorder. In most cases, ADPKD similarly affects bilateral kidneys. CASE PRESENTATION: Among the 605 ADPKD patients that were followed up by our center, we identified two male patients with unilateral ADPKD. The cases were remarkable because the patients also had ectopia and multicystic dysplasia in the contralateral kidney, which are generally sporadic disease conditions. Both patients tested positive for polycystic kidney disease 1 mutation, but negative for hepatocyte nuclear factor 1 beta mutation. Moreover, the deterioration of their kidney function seemed to be quicker than their age- and sex-matched controls and siblings. Both patients had started a long-term hemodialysis in their 40s.
conclusionAnatomical and genetic abnormality in patients with ADPKD may be more frequent and complex than previously believed. The compensatory capacity in patients with ADPKD is fragile, and missing one kidney could accelerate the deterioration of renal function.
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