ArticleScientific reports2013
Feedback mechanism in depolarization-induced sustained activation of extracellular signal-regulated kinase in the hippocampus.
Article in Scientific reports, 2013. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 16 papers.
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Who cites it
16 citing papers in PubMed, 23 citations in OpenAlex.
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- Neurosteroids Alter p-ERK Levels and Tau Distribution, Restraining the Effects of High Extracellular Calcium.International journal of molecular sciences · 2024Article
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- Precise timing of ERK phosphorylation/dephosphorylation determines the outcome of trial repetition during long-term memory formation.Proceedings of the National Academy of Sciences of the United States of America · 2022Article
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- Computational model of a positive BDNF feedback loop in hippocampal neurons following inhibitory avoidance training.Learning & memory (Cold Spring Harbor, N.Y.) · 2016Article
- Caveolin-1 regulates corneal wound healing by modulating Kir4.1 activity.American journal of physiology. Cell physiology · 2016Article
- A PARP1-ERK2 synergism is required for the induction of LTP.Scientific reports · 2016Article
- Dopamine D1/D5 receptor signaling regulates synaptic cooperation and competition in hippocampal CA1 pyramidal neurons via sustained ERK1/2 activation.Hippocampus · 2016Article
- A PARP1-Erk2 synergism is required for stimulation-induced expression of immediate early genes.Gene & translational bioinformatics · 2016Article
- Tanshinone IIA Alleviates the AD Phenotypes in APP and PS1 Transgenic Mice.BioMed research international · 2016Article
- MPTP-induced changes in hippocampal synaptic plasticity and memory are prevented by memantine through the BDNF-TrkB pathway.British journal of pharmacology · 2015Article
- Depolarization of Cellular Resting Membrane Potential Promotes Neonatal Cardiomyocyte Proliferation In Vitro.Cellular and molecular bioengineering · 2014Article
- Dab1 is required for synaptic plasticity and associative learning.The Journal of neuroscience : the official journal of the Society for Neuroscience · 2013Article
- REV, A BRET-Based Sensor of ERK Activity.Frontiers in endocrinology · 2013Article
Corrections and comments
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Authors and funding
3 authors at 2 institutions in 2 countries.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Phosphorylation plays important roles in several processes including synaptic plasticity and memory. The critical role of extracellular signal-regulated kinase (ERK) in these processes is well established. ERK is activated in a sustained manner by different stimuli. However, the mechanisms of sustained ERK activation are not completely understood. Here we show that KCl depolarization-induced sustained ERK activation in the hippocampal slices is critically dependent on protein synthesis and transcription. In addition, the sustained ERK activation requires receptor tyrosine kinase(s) activity. In support of a role for a growth factor in sustained ERK activation, KCl depolarization enhances the level of brain-derived neurotrophic factor (BDNF). Furthermore, BDNF antibody blocks KCl-induced sustained ERK activation. These results suggest a positive feed-back loop in which depolarization-induced BDNF maintains ERK activation in the sustained phase.
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