Evidence map›Paper›PMID 23336732›Full record

ReviewBritish journal of pharmacology2013

Modulation of neutrophil oxidative burst via histamine receptors.

M Cíž, A Lojek

Abstract readReview
In one paragraph

Review in British journal of pharmacology, 2013. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 15 papers.

0numbers the graph read from it
0cells of the map it votes in
15citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

15 citing papers in PubMed.

  1. Article
  2. Article
  3. Review
  4. Medical Management of COVID-19: Evidence and Experience.Journal of clinical medicine research · 2020
    Review
  5. Article
  6. Observational
  7. Review
  8. Review
  9. Aged Lymphatic Vessels and Mast Cells in Perilymphatic Tissues.International journal of molecular sciences · 2017
    Review
  10. Article
  11. Article
  12. Article
  13. Article
  14. Review
  15. Histamine pharmacology: four years on.British journal of pharmacology · 2013
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

M CížDepartment of Free Radical Pathophysiology, Institute of Biophysics, Academy of Sciences of the Czech Republic, Brno, Czech Republic. milanciz@ibp.cz
A Lojek

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Histamine has the ability to influence the activity of immune cells including neutrophils and plays a pivotal role in inflammatory processes, which are a complex network of cellular and humoral events. One of the main functions manifested by activated neutrophils is oxidative burst, which is linked to the production of reactive oxygen species; therefore, the effects of histamine receptor agonists and antagonists on the oxidative burst of neutrophils is reviewed. A role for the well-characterized histamine H1 and H2 receptors in this process is discussed and compared to that of the recently discovered H4 receptor.

Indexed as

AnimalsHistamineHistamine AgonistsHistamine AntagonistsHumansInflammationNeutrophilsReactive Oxygen SpeciesReceptors, G-Protein-CoupledReceptors, HistamineReceptors, Histamine H1Receptors, Histamine H2Receptors, Histamine H4Respiratory BurstHistamineHistamine AgonistsHistamine AntagonistsHRH4 protein, humanReactive Oxygen SpeciesReceptors, G-Protein-CoupledReceptors, HistamineReceptors, Histamine H1Receptors, Histamine H2Receptors, Histamine H4histaminehistamine receptorneutrophiloxidative burstreactive oxygen species

Identifiers

PMID23336732
PMCPMC3764845

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.