ArticleJournal of emergencies, trauma, and shock2012
Rare combination of bilateral putaminal necrosis, optic neuritis, and polyneuropathy in a case of acute methanol intoxication among patients met with hooch tragedy in Gujarat, India.
Article in Journal of emergencies, trauma, and shock, 2012. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.
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5 citing papers in PubMed, 8 citations in OpenAlex.
- Biochemical mechanism underlying the pathogenesis of diabetic retinopathy and other diabetic complications in humans: the methanol-formaldehyde-formic acid hypothesis.Acta biochimica et biophysica Sinica · 2022Article
- Methanol poisoning as a new world challenge: A review.Annals of medicine and surgery (2012) · 2021Review
- Effects of methanol and formic acid on human platelet aggregation.Environmental health and preventive medicine · 2017Article
- Neuromuscular Functions on Experimental Acute Methanol Intoxication.Turkish journal of anaesthesiology and reanimation · 2015Article
- Multifocal neuraxial involvement in acute methanol intoxication: A series of two patients from rural India.Neurology perspectivesArticle
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4 authors at 2 institutions in 1 country.
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Abstract
Methanol poisoning is a rare but extremely hazardous form of intoxication, generally occurring after suicidal or accidental events. Methanol is a cheap and potent adulterant of illicit liquors. In India, we have witnessed number of mass emergencies due to adulterated alcohol consumption. Although Gujarat State had banned alcohol consumption since 1961, worse hooch tragedies have often taken place. The most severe consequences of methanol intoxication are blindness, a profound metabolic acidosis and various forms of neurological impairment; which occur characteristically after a latent period of several hours or days after ingestion. We present a unique case of acute methanol intoxication presented with, apart from metabolic acidosis and optic neuritis, involvement of central nervous system and peripheral nervous system. He had bilateral optic neuritis, delayed onset polyneuropathy with axonopathy, and radiculopathy. Magnetic resonance imaging findings were consistent with bilateral putaminal necrosis.
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