Evidence map›Paper›PMID 23241323›Full record

ArticleAmerican journal of physiology. Heart and circulatory physiology2013

Glucagon-like peptide-1 preserves coronary microvascular endothelial function after cardiac arrest and resuscitation: potential antioxidant effects.

Betsy B Dokken, Charles V Piermarini, Mary K Teachey, Michael T Gura, Christian J Dameff, Brian D Heller, Jonida Krate, Aeen M Ashgar, Lauren Querin, Jennifer L Mitchell and 2 more

Erratum issued Registry-linked trialAbstract read
PubMed Publisher
In one paragraph

Article in American journal of physiology. Heart and circulatory physiology, 2013. The graph could read no effect estimate from its abstract, so it casts no vote on the map. An erratum has been issued. It is linked to trial NCT02621489 (Effects on Re-endothelialisation With Bydureon Treatment Add on to Insulin Versus Insulin Alone, Both in Combination With Metformin in Type 2 Diabetic Subjects), which is not on this map. Cited by 15 papers.

0numbers the graph read from it
0cells of the map it votes in
15citing papers in PubMed
4.4field-weighted citation impact, top 6% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

NCT02621489 phase4completedstarted 2015, after this paper: background citation

Effects on Re-endothelialisation With Bydureon Treatment Add on to Insulin Versus Insulin Alone, Both in Combination With Metformin in Type 2 Diabetic Subjects

Ran2015Enrolled38Registered outcomes16Posted comparisons0ConditionsAtherosclerosis, Diabetes, RestenosisArmsBydureon, Humulin kwickpen, Metformin
Open the trial in the graph
3 · Its place in the literature

Who cites it

15 citing papers in PubMed, 30 citations in OpenAlex.

  1. Trial
  2. Trial
  3. Review
  4. Review
  5. Observational
  6. Review
  7. Glucagon-like Peptide-1 Secretion Is Inhibited by Lysophosphatidic Acid.International journal of molecular sciences · 2022
    Article
  8. Article
  9. Linagliptin treatment improves cerebrovascular function and remodeling and restores reduced cerebral perfusion in Type 2 diabetes.American journal of physiology. Regulatory, integrative and comparative physiology · 2016
    Article
  10. Review
  11. Review
  12. Review
  13. Incretin attenuates diabetes-induced damage in rat cardiac tissue.The journal of physiological sciences : JPS · 2014
    Article
  14. Article
  15. Direct cardiovascular effects of glucagon like peptide-1.Diabetology & metabolic syndrome · 2013
    Article
4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

12 authors at 1 institution in 1 country.

Betsy B DokkenDepartment of Medicine, University of Arizona, Tucson, AZ, USA. bdokken@deptofmed.arizona.edu
Charles V Piermarini
Mary K Teachey
Michael T Gura
Christian J Dameff
Brian D Heller
Jonida Krate
Aeen M Ashgar
Lauren Querin
Jennifer L Mitchell
Ronald W Hilwig
Karl B Kern
University of Arizona · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Glucagon-like peptide-1 (GLP-1) has protective effects in the heart. We hypothesized that GLP-1 would mitigate coronary microvascular and left ventricular (LV) dysfunction if administered after cardiac arrest and resuscitation (CAR). Eighteen swine were subjected to ventricular fibrillation followed by resuscitation. Swine surviving to return of spontaneous circulation (ROSC) were randomized to receive an intravenous infusion of either human rGLP-1 (10 pmol·kg(-1)·min(-1); n = 8) or 0.9% saline (n = 8) for 4 h, beginning 1 min after ROSC. CAR caused a decline in coronary flow reserve (CFR) in control animals (pre-arrest, 1.86 ± 0.20; 1 h post-ROSC, 1.3 ± 0.05; 4 h post-ROSC, 1.25 ± 0.06; P < 0.05). GLP-1 preserved CFR for up to 4 h after ROSC (pre-arrest, 1.31 ± 0.17; 1 h post-ROSC, 1.5 ± 0.01; 4 h post-ROSC, 1.55 ± 0.22). Although there was a trend toward improvement in LV relaxation in the GLP-1-treated animals, overall LV function was not consistently different between groups. 8-iso-PGF(2α), a measure of reactive oxygen species load, was decreased in post-ROSC GLP-1-treated animals [placebo, control (NS): 38.1 ± 1.54 pg/ml; GLP-1: 26.59 ± 1.56 pg/ml; P < 0.05]. Infusion of GLP-1 after CAR preserved coronary microvascular and LV diastolic function. These effects may be mediated through a reduction in oxidative stress.

Indexed as

Cardiopulmonary ResuscitationAnimalsAntioxidantsCoronary CirculationDinoprostEndothelium, VascularFemaleGlucagon-Like Peptide 1Heart ArrestHeart VentriclesHumansMaleMicrovesselsReactive Oxygen SpeciesSwineVentricular Dysfunction, Left8-epi-prostaglandin F2alphaAntioxidantsDinoprostGlucagon-Like Peptide 1Reactive Oxygen Species

Identifiers

PMID23241323
OpenAlexW2113328186

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.