Editorial: More evidence for a role of CpG methylation in the pathogenesis of osteoarthritis.

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

Who cites it

2 citing papers in PubMed.

1. Underlying molecular mechanisms of DIO2 susceptibility in symptomatic osteoarthritis.Annals of the rheumatic diseases · 2015
   Article
2. Genome-wide mapping of DNA hydroxymethylation in osteoarthritic chondrocytes.Arthritis & rheumatology (Hoboken, N.J.) · 2015
   Article

Corrections and comments

• Comment on
  Loss of methylation in CpG sites in the NF-κB enhancer elements of inducible nitric oxide synthase is responsible for gene induction in human articular chondrocytes.2013

Authors and funding

1 author.

Funding

Abstract

PubMed holds no abstract for this paper.

Indexed as