ReviewCancer science2013
Epithelial mesenchymal transition from a natural gestational orchestration to a bizarre cancer disturbance.
Review in Cancer science, 2013. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 15 papers.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
15 citing papers in PubMed, 26 citations in OpenAlex.
- The Expression Analysis ofInternational journal of hematology-oncology and stem cell research · 2024Article
- Single-cell transcriptomic analysis reveals the critical molecular pattern of UV-induced cutaneous squamous cell carcinoma.Cell death & disease · 2021Article
- Differential expression ofEXCLI journal · 2020Article
- Article
- Differential Expression Pattern of Epithelial Mesenchymal Transition Gens: AXL, GAS6, Claudin-1, and Cofilin-1, in Different Stages of Epithelial Ovarian Cancer.Iranian journal of public health · 2019Article
- Article
- Fibroblast growth factor-10 and epithelial-mesenchymal transition in colorectal cancer.EXCLI journal · 2019Article
- Silencing NUDT21 Attenuates the Mesenchymal Identity of Glioblastoma Cells via the NF-κB Pathway.Frontiers in molecular neuroscience · 2017Article
- MicroRNA-182 targets SMAD7 to potentiate TGFβ-induced epithelial-mesenchymal transition and metastasis of cancer cells.Nature communications · 2016Article
- Conditioned Medium from Adipose-Derived Stem Cells (ADSCs) Promotes Epithelial-to-Mesenchymal-Like Transition (EMT-Like) in Glioma Cells In vitro.Molecular neurobiology · 2016Article
- Serine/Threonine Kinase MLK4 Determines Mesenchymal Identity in Glioma Stem Cells in an NF-κB-dependent Manner.Cancer cell · 2016Article
- Rac1 expression in epithelial ovarian cancer: effect on cell EMT and clinical outcome.Medical oncology (Northwood, London, England) · 2015Article
- 3,3'-Diindolylmethane inhibits the invasion and metastasis of nasopharyngeal carcinoma cellsExperimental and therapeutic medicine · 2014Article
- New insights into the morphogenic role of stromal cells and their relevance for regenerative medicine. lessons from the heart.Journal of cellular and molecular medicine · 2014Review
- FGF10: Type III Epithelial Mesenchymal Transition and Invasion in Breast Cancer Cell Lines.Journal of Cancer · 2014Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
1 author at 1 institution in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
The epithelial to mesenchymal transition (EMT), a pathologic phenomenon in cancer, has a twin in the embryonic period of life. In the first one, its promotion will cause metastasis to become a life-threatening stage of cancer, while in the second it will lead to organogenesis, which is necessary for all living creatures. There is one more from this phenomenon, which occurs during the wound healing process and if dys-regulated can lead to fibrosis. In both there are stimulants in common and one that are different. Stages start from cell-cell junction dissociation followed by morphological changes and behavioral and essence alterations. To control the EMT as a bizarre disturbance in cancer and metastasis, initially it is better to understand the wonder of natural gestational orchestration in early life. In this review, first the structure of the two heads of the spectrum is described followed by the cellular and micro-environmental alterations during this phenomenon. Understanding cellular behavior in this process and what makes them invasive resistant stemness cells will be of great importance in highlighting roads to cancer treatment.
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.