ArticleJournal of neurochemistry2013

Targeted deletion of the antisilencer/enhancer (ASE) element from intron 1 of the myelin proteolipid protein gene (Plp1) in mouse reveals that the element is dispensable for Plp1 expression in brain during development and remyelination.

Glauber B Pereira et al.PubMed ↗Full text ↗Publisher ↗

No numbers read from the abstract.