Evidence map›Paper›PMID 23135745›Full record

ArticleGenetic epidemiology2013

Trees Assembling Mann-Whitney approach for detecting genome-wide joint association among low-marginal-effect loci.

Changshuai Wei, Daniel J Schaid, Qing Lu

Open access · greenAbstract read
In one paragraph

Article in Genetic epidemiology, 2013. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers.

0numbers the graph read from it
0cells of the map it votes in
8citing papers in PubMed
1.4field-weighted citation impact, top 16% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

8 citing papers in PubMed, 10 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 2 institutions in 1 country.

Changshuai WeiDepartment of Epidemiology and Biostatistics, Michigan State University, East Lansing, Michigan 48824, USA.
Daniel J Schaid
Qing Lu
Michigan State University · USMayo Clinic · US

Funding

Quantitative Methods for Genetic EpidemiologyR01GM065450 · NIGMS · MAYO CLINIC ROCHESTER · PI SCHAID, DANIEL J. · 2002 to 2020
$5.6M
Gene-Gene/Gene-Environment Interactions Associated with Nicotine DependenceK01DA033346 · NIDA · MICHIGAN STATE UNIVERSITY · PI LU, QING · 2013 to 2017
$833k
High-dimensional Statistical Genetic Approach for Family-based Orofacial CleftsR03DE022379 · NIDCR · MICHIGAN STATE UNIVERSITY · PI LU, QING · 2012 to 2013
$441k
NIDA NIH HHS K01 DA033346NIDCR NIH HHS R03 DE022379NIDCR NIH HHS R03DE022379NIGMS NIH HHS R01 GM065450Wellcome Trust
6 · The paper itself

Abstract

Common complex diseases are likely influenced by the interplay of hundreds, or even thousands, of genetic variants. Converging evidence shows that genetic variants with low marginal effects (LMEs) play an important role in disease development. Despite their potential significance, discovering LME genetic variants and assessing their joint association on high-dimensional data (e.g., genome-wide data) remain a great challenge. To facilitate joint association analysis among a large ensemble of LME genetic variants, we proposed a computationally efficient and powerful approach, which we call Trees Assembling Mann-Whitney (TAMW). Through simulation studies and an empirical data application, we found that TAMW outperformed multifactor dimensionality reduction (MDR) and the likelihood ratio-based Mann-Whitney approach (LRMW) when the underlying complex disease involves multiple LME loci and their interactions. For instance, in a simulation with 20 interacting LME loci, TAMW attained a higher power (power = 0.931) than both MDR (power = 0.599) and LRMW (power = 0.704). In an empirical study of 29 known Crohn's disease (CD) loci, TAMW also identified a stronger joint association with CD than those detected by MDR and LRMW. Finally, we applied TAMW to Wellcome Trust CD GWAS to conduct a genome-wide analysis. The analysis of 459K single nucleotide polymorphisms was completed in 40 hrs using parallel computing, and revealed a joint association predisposing to CD (P-value = 2.763 × 10(-19)). Further analysis of the newly discovered association suggested that 13 genes, such as ATG16L1 and LACC1, may play an important role in CD pathophysiological and etiological processes.

Indexed as

Genetic LociGenetic Predisposition to DiseaseGenome-Wide Association StudyModels, GeneticPolymorphism, Single NucleotideComputer SimulationCrohn DiseaseHumansMultifactor Dimensionality Reduction

Identifiers

PMID23135745
PMCPMC3593719
OpenAlexW2949747779

What OpenQuestion holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.