Evidence map›Paper›PMID 23076393›Full record

ArticleMammalian genome : official journal of the International Mammalian Genome Society2013

Combined gene dosage requirement for SWI/SNF catalytic subunits during early mammalian development.

Stephanie L Smith-Roe, Scott J Bultman

Abstract read
In one paragraph

Article in Mammalian genome : official journal of the International Mammalian Genome Society, 2013. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 14 papers.

0numbers the graph read from it
0cells of the map it votes in
14citing papers in PubMed
0.8field-weighted citation impact, top 29% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

14 citing papers in PubMed, 23 citations in OpenAlex.

  1. Characterization ofBiomolecules · 2026
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  14. SWI/SNF chromatin-remodeling complexes in cardiovascular development and disease.Cardiovascular pathology : the official journal of the Society for Cardiovascular Pathology
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors at 2 institutions in 1 country.

Stephanie L Smith-RoeDepartment of Genetics, University of North Carolina, Chapel Hill, NC 27599-7264, USA.
Scott J Bultman
UNC Lineberger Comprehensive Cancer CenterUniversity of North Carolina at Chapel Hill · US

Funding

Dietary Fiber, Gut Microflora, & Epigenetic Events that Prevent Colorectal CancerR01CA125237 · NCI · UNIV OF NORTH CAROLINA CHAPEL HILL · PI BULTMAN, SCOTT J · 2007 to 2011
$1.4M
NCI NIH HHS CA125237NCI NIH HHS R01 CA125237
6 · The paper itself

Abstract

Mammalian SWI/SNF complexes utilize either BRG1 or BRM as alternative catalytic subunits with DNA-dependent ATPase activity to remodel chromatin. Although the two proteins are 75 % identical, broadly expressed, and have similar biochemical activities in vitro, BRG1 is essential for mouse embryonic development, while BRM is dispensable. To investigate whether BRG1 and BRM have overlapping functions during mouse embryogenesis, we performed double-heterozygous intercrosses using constitutive null mutations previously created by gene targeting. The progeny of these crosses had a distribution of genotypes that was significantly skewed relative to their combined gene dosage. This was most pronounced at the top and bottom of the gene dosage hierarchy, with a 1.5-fold overrepresentation of Brg1 (+/+) ;Brm (+/+) mice and a corresponding 1.6-fold underrepresentation of Brg1 (+/-) ;Brm (-/-) mice. To account for the underrepresentation of Brg1 (+/-) ;Brm (-/-) mice, timed matings and blastocyst outgrowth assays demonstrated that ~50 % of these embryos failed to develop beyond the peri-implantation stage. These results challenge the idea that BRG1 is the exclusive catalytic subunit of SWI/SNF complexes in ES cells and suggest that BRM also interacts with the pluripotency transcription factors to facilitate self-renewal of the inner cell mass. In contrast to implantation, the Brm genotype did not influence an exencephaly phenotype that arises because of Brg1 haploinsufficiency during neural tube closure and that results in peri-natal lethality. Taken together, these results support the idea that BRG1 and BRM have overlapping functions for certain developmental processes but not others during embryogenesis.

Indexed as

Gene DosageAdenosine TriphosphatasesAllelesAnimalsBlotting, WesternCatalytic DomainCells, CulturedChromatin Assembly and DisassemblyDNA HelicasesEmbryonic DevelopmentFibroblastsGenotypeMiceNuclear ProteinsPhenotypeTranscription FactorsAdenosine TriphosphatasesDNA HelicasesNuclear ProteinsSmarca2 protein, mouseSmarca4 protein, mouseTranscription Factors

Identifiers

PMID23076393
PMCPMC3562406
OpenAlexW2035495088

What OpenQuestion holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.