ArticleJournal of bacteriology2012
Identification and characterization of the functional toxboxes in the Vibrio cholerae cholera toxin promoter.
Article in Journal of bacteriology, 2012. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 12 papers.
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Who cites it
12 citing papers in PubMed, 25 citations in OpenAlex.
- Strain-dependentFrontiers in microbiology · 2026Article
- H-NS and ToxT Inversely Control Cholera Toxin Production by Binding to Overlapping DNA Sequences.Journal of bacteriology · 2021Article
- Deciphering the possible role of ctxB7 allele on higher production of cholera toxin by Haitian variant Vibrio cholerae O1.PLoS neglected tropical diseases · 2020Article
- Targeting of cholera toxin A (Research in pharmaceutical sciences · 2020Article
- Structural basis for virulence regulation inCommunications biology · 2019Article
- Conjugated Linoleic Acid Reduces Cholera Toxin Production In Vitro and In Vivo by Inhibiting Vibrio cholerae ToxT Activity.Antimicrobial agents and chemotherapy · 2015Article
- Mechanism for inhibition of Vibrio cholerae ToxT activity by the unsaturated fatty acid components of bile.Journal of bacteriology · 2015Article
- A small unstructured region in Vibrio cholerae ToxT mediates the response to positive and negative effectors and ToxT proteolysis.Journal of bacteriology · 2015Article
- Bicarbonate increases binding affinity of Vibrio cholerae ToxT to virulence gene promoters.Journal of bacteriology · 2014Article
- Zebrafish as a natural host model for Vibrio cholerae colonization and transmission.Applied and environmental microbiology · 2014Article
- Article
- ToxR recognizes a direct repeat element in the toxT, ompU, ompT, and ctxA promoters of Vibrio cholerae to regulate transcription.Infection and immunity · 2013Article
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Authors and funding
2 authors at 1 institution in 1 country.
Funding
Abstract
Following the consumption of contaminated food or water by a human host, the Vibrio cholerae bacterium produces virulence factors, including cholera toxin (CT), which directly causes voluminous diarrhea, producing cholera. A complex regulatory network controls virulence gene expression and responds to various environmental signals and transcription factors. Ultimately, ToxT, a member of the AraC/XylS transcription regulator family, is responsible for activating the transcription of the virulence genes. ToxT-regulated promoters all contain one or more copies of the toxbox, a 13-bp DNA sequence which ToxT recognizes. Nucleotides 2 through 7 of the toxbox sequence are well conserved and contain an invariant tract of four consecutive T nucleotides, whereas the remainder of the toxbox sequence is not highly conserved other than being A/T rich. The binding of ToxT to toxboxes is required to activate the transcription of virulence genes, and toxboxes in several virulence gene promoters have been characterized. However, the toxboxes required for the activation of transcription from the cholera toxin promoter PctxAB have not been identified. PctxAB contains a series of heptad repeats (GATTTTT), each of which matches the 5' end of the toxbox consensus sequence and is a potential binding site for ToxT. Using site-directed mutagenesis and high-resolution copper-phenanthroline footprinting, we have identified the functional toxboxes required for the ToxT activation of PctxAB. Our findings suggest that ToxT binds to only two toxboxes within PctxAB, despite the presence of several other potential ToxT binding sites within the promoter. Both toxboxes are essential for DNA binding and the full activation of ctxAB transcription.
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