Evidence map›Paper›PMID 22739230›Full record

ArticleAmerican journal of kidney diseases : the official journal of the National Kidney Foundation2012

Subclinical celiac disease and crystal-induced kidney disease following kidney transplant.

Giovanna Capolongo, Sameh Abul-Ezz, Orson W Moe, Khashayar Sakhaee

Abstract read
In one paragraph

Article in American journal of kidney diseases : the official journal of the National Kidney Foundation, 2012. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 16 papers.

0numbers the graph read from it
0cells of the map it votes in
16citing papers in PubMed
0.6field-weighted citation impact, top 29% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

16 citing papers in PubMed, 27 citations in OpenAlex.

  1. Review
  2. Article
  3. Review
  4. Article
  5. Article
  6. Oxalate homeostasis.Nature reviews. Nephrology · 2023
    Review
  7. A rare cause of nephrocalcinosis in an infant: Answers.Pediatric nephrology (Berlin, Germany) · 2021
    Article
  8. Article
  9. The association of calcium oxalate deposition in kidney allografts with graft and patient survival.Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association · 2020
    Article
  10. Secondary Oxalate Nephropathy: A Systematic Review.Kidney international reports · 2018
    Article
  11. Article
  12. Article
  13. Article
  14. Observational
  15. Loss of Cystic Fibrosis Transmembrane Regulator Impairs Intestinal Oxalate Secretion.Journal of the American Society of Nephrology : JASN · 2017
    Article
  16. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 2 institutions in 1 country.

Giovanna CapolongoCharles & Jane Pak Center for Mineral Metabolism & Clinical Research, University of Texas Southwestern Medical Center, Dallas, USA.
Sameh Abul-Ezz
Orson W Moe
Khashayar Sakhaee
The University of Texas Southwestern Medical Center · USUniversity of Arkansas for Medical Sciences · US

Funding

XU 40468 IN HYPERCHOLESTEROLEMIAM01RR000633 · NCRR · UT SOUTHWESTERN MEDICAL CENTER · PI TOTO, ROBERT DANIEL · 1985 to 2007
$21.0M
RENAL BASIS OF HYPOCITRATURIAP01DK020543 · NIDDK · UT SOUTHWESTERN MEDICAL CENTER · PI HUANG, CHOU-LONG · 1986 to 2008
$12.7M
Pathogenesis of Uric Acid Nephrolithiasis: The Multifaceted Role of Renal LipidsR01DK081423 · NIDDK · UT SOUTHWESTERN MEDICAL CENTER · PI MAALOUF, NAIM M, MOE, ORSON W · 2009 to 2023
$6.1M
NCRR NIH HHS M01 RR000633NCRR NIH HHS M01-RR00633NIDDK NIH HHS P01 DK020543NIDDK NIH HHS P01-DK20543NIDDK NIH HHS R01 DK081423NIDDK NIH HHS R01-DK081423
6 · The paper itself

Abstract

Decreased kidney function from kidney deposition of calcium oxalate has been described previously in inflammatory bowel disease and after jejuno-ileal and Roux-en-Y gastric bypass surgeries. Although celiac disease is the most prevalent bowel abnormality associated with intestinal malabsorption, its relationship to high kidney oxalate burden and decreased kidney function has not been established. We report a case of subclinical celiac disease and hyperoxaluria that presented with loss of kidney function as a result of high oxalate load in the absence of overt diarrhea, documented intestinal fat malabsorption, and nephrolithiasis. Subclinical celiac disease is commonly overlooked and hyperoxaluria is not usually investigated in kidney patients. We propose that this entity should be suspected in patients with chronic kidney disease in which the cause of kidney damage has not been clearly established.

Indexed as

Kidney TransplantationCeliac DiseaseCreatinineFemaleHumansHyperoxaluriaImmunohistochemistryKidney DiseasesKidney TubulesMalabsorption SyndromesMembrane Transport ProteinsMiddle AgedOxalatesSulfate TransportersCreatinineMembrane Transport ProteinsOxalatesSLC26A6 protein, humanSulfate Transporters

Identifiers

PMID22739230
PMCPMC4469185
OpenAlexW2016544862

What OpenQuestion holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.