ArticleInternational journal of clinical and experimental medicine2012
Role of endogenous testosterone in TNF-induced myocardial injury in males.
Article in International journal of clinical and experimental medicine, 2012. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.
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Who cites it
5 citing papers in PubMed, 11 citations in OpenAlex.
- Article
- Androgen Signaling Regulates SARS-CoV-2 Receptor Levels and Is Associated with Severe COVID-19 Symptoms in Men.Cell stem cell · 2020Article
- Article
- Androgen Regulates SARS-CoV-2 Receptor Levels and Is Associated with Severe COVID-19 Symptoms in Men.bioRxiv : the preprint server for biology · 2020Article
- Tumor necrosis factor-alpha induces VCAM-1-mediated inflammation via c-Src-dependent transactivation of EGF receptors in human cardiac fibroblasts.Journal of biomedical science · 2015Article
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Authors and funding
4 authors at 1 institution in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
backgroundGender-specific disparities have been observed in myocardium exposed to tumor necrosis factor-α (TNF). Male myocardium demonstrates greater loss in cardiac function in the presence of a given TNF level compared to female. In addition, we have previously demonstrated that estrogen has little influence on reducing TNF-caused myocardial dysfunction in female hearts, suggesting that male hormone - testosterone may be responsible for gender differences in TNF-mediated myocardial damage. Therefore, in this study, we hypothesize that endogenous testosterone plays a detrimental role in TNF-induced myocardial injury in male hearts.
methodsIsolated mouse hearts from age-matched adult males, females, castrated males and males treated with androgen receptor blocker-flutamide were subjected to 45 minutes of TNF infusion via a Langendorff model. Left ventricular developed pressure (LVDP) and heart rate were continuously recorded. After TNF infusion, heart tissue was analyzed for myocardial levels of caspase-8 and caspase-3 by Western blot assay.
resultsTNF infusion significantly depressed LVDP, but not heart rate in males. Myocardial rate pressure product (RPP, LVDP*heart rate) was markedly decreased in male hearts compared to females in exposure to TNF, which was associated with higher levels of TNF-induced caspase-8 and caspase-3. Importantly, depletion of endogenous testosterone by castration or blockade of androgen receptor by flutamide treatment abolished TNF-decreased RPP in male hearts. However, castration or flutamide treatment did not affect TNF production and myocardial expression of TNFR1 and TNFR2.
conclusionOur study shows that testosterone is critical to the gender difference in TNF-induced detrimental effects on myocardium. Relative low threshold for TNF-caused myocardial damage in males is likely due to the interaction of testosterone with downstream signals of TNFR1 and/or TNFR2.
Indexed as
Identifiers
22567171PMC3342716W195124802What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.