ArticleExperimental diabetes research2012
Inflammation, diabetes, and chronic kidney disease: role of aerobic capacity.
Article in Experimental diabetes research, 2012. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.
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Who cites it
6 citing papers in PubMed.
- Associations of composite dietary antioxidant index with suicidal ideation incidence and mortality among the U.S. population.Frontiers in nutrition · 2024Article
- Assessment of the Phenolic Profiles, Hypoglycemic Activity, and Molecular Mechanism of Different Highland Barley (International journal of molecular sciences · 2020Article
- Development and validation of a Fall Risk Assessment Index for dialysis patients.Clinical and experimental nephrology · 2018Article
- Systemic Inflammation in Midlife: Race, Socioeconomic Status, and Perceived Discrimination.American journal of preventive medicine · 2017Observational
- Tumor necrosis factor alpha -238 G/A and -308 G/A polymorphisms and soluble TNF-α levels in chronic kidney disease: correlation with clinical variables.International journal of clinical and experimental medicine · 2014Article
- Inflammatory factors and exercise in chronic kidney disease.International journal of endocrinology · 2013Article
Corrections and comments
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Authors and funding
9 authors.
Funding
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Abstract
The persistent inflammatory state is common in diabetes and chronic kidney disease (CKD). These patients present exercise intolerance and increased arterial stiffness. Long-term aerobic exercise has been associated with better arterial compliance, antidiabetic and antiinflammatory benefits. We assessed the hypothesis that in patients with diabetes and CKD, better aerobic capacity is associated with less inflammatory state and arterial stiffness. Thirty-nine CKD patients (17 in hemodialysis) were evaluated. According to CKD etiology two patient groups were obtained: group of diabetics (GD) was formed by 11 patients and nondiabetics (GND) formed by 28 patients. Central blood pressure and arterial stiffness were evaluated by Sphygmocor device. Carotida intima-media thickness (CA-IMT) was evaluated by ultrasonography. Aerobic capacity was measured by estimated VO(2)max according to treadmill test by Bruce protocol. The GD showed a higher frequency of C-reactive protein above laboratory cutoff (P = 0.044), higher frequency of male gender, and a non significant higher value of VO(2)max (P = 0.099). The CA-IMT was similar. Only better aerobic capacity was associated with lower frequency of high C-reactive protein when adjusted to diabetes and gender in a logistic regression model. In conclusion, aerobic capacity was associated with inflammatory state, in CKD patients, independently of diabetes presence.
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