Evidence map›Paper›PMID 22548961›Full record

ReviewBiochimica et biophysica acta2013

The meaning of mitochondrial movement to a neuron's life.

Jonathan R Lovas, Xinnan Wang

Abstract readReview
In one paragraph

Review in Biochimica et biophysica acta, 2013. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 35 papers.

0numbers the graph read from it
0cells of the map it votes in
35citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

35 citing papers in PubMed.

  1. Article
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  8. Review
  9. Neurohormonal connections with mitochondria in cardiomyopathy and other diseases.American journal of physiology. Cell physiology · 2022
    Review
  10. Article
  11. Article
  12. Review
  13. Article
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  15. Article
  16. Review
  17. Article
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  20. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Jonathan R LovasDepartment of Neurosurgery, Stanford University School of Medicine, USA. lovas@stanford.edu
Xinnan Wang

Funding

Mis-regulation of Mitochondrial Motility in Parkinsonian NeurodegenerationR00NS067066 · NINDS · STANFORD UNIVERSITY · PI WANG, XINNAN · 2012 to 2014
$729k
NINDS NIH HHS R00 NS067066PHS HHS R0O067066
6 · The paper itself

Abstract

Cells precisely regulate mitochondrial movement in order to balance energy needs and avoid cell death. Neurons are particularly susceptible to disturbance of mitochondrial motility and distribution due to their highly extended structures and specialized function. Regulation of mitochondrial motility plays a vital role in neuronal health and death. Here we review the current understanding of regulatory mechanisms that govern neuronal mitochondrial transport and probe their implication in health and disease. This article is part of a Special Issue entitled: Mitochondrial dynamics and physiology.

Indexed as

AnimalsBiological TransportDiseaseHumansMitochondriaMitochondrial DynamicsModels, BiologicalMovementNeurons

Identifiers

PMID22548961
PMCPMC3413748

What OpenQuestion holds

Textmetadata
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.