Evidence map›Paper›PMID 22515108›Full record

ReviewNagoya journal of medical science2012

Adipocytokines and obesity-linked disorders.

Noriyuki Ouchi, Koji Ohashi, Rei Shibata, Toyoaki Murohara

Abstract readReview
In one paragraph

Review in Nagoya journal of medical science, 2012. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 33 papers.

0numbers the graph read from it
0cells of the map it votes in
33citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

33 citing papers in PubMed.

  1. Article
  2. Review
  3. The Interplay between Obesity and Inflammation.Life (Basel, Switzerland) · 2024
    Review
  4. Recent progress in adipocytokine research.Nagoya journal of medical science · 2023
    Article
  5. Article
  6. Revealing Potential Bioactive Compounds and Mechanisms ofCurrent issues in molecular biology · 2022
    Article
  7. The Effect of Garlic (Clinical nutrition research · 2021
    Article
  8. Article
  9. Review
  10. The Role of microRNAs in Metabolic Syndrome-Related Oxidative Stress.International journal of molecular sciences · 2020
    Review
  11. Review
  12. Obesity and chronic inflammation crosslinking.Central-European journal of immunology · 2020
    Review
  13. Article
  14. Article
  15. Article
  16. Article
  17. Article
  18. Article
  19. Association of circulating omentin-1 level with lung cancer in smokers.Medical journal of the Islamic Republic of Iran · 2018
    Article
  20. Review
4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

4 authors.

Noriyuki OuchiDepartment of Molecular Cardiology, Nagoya University Graduate School of Medicine, 65 Tsurumai-cho Showa-ku, Nagoya, 466-8550, Japan. nouchi@med.nagoya-u.ac.jp
Koji Ohashi
Rei Shibata
Toyoaki Murohara

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Obesity is closely associated with an increased risk for metabolic and cardiovascular diseases. Adipose tissue produces a number of secretory bioactive substances, also known as adipocytokines or adipokines, which directly affect adjacent or distant organs. Most adipocytokines are pro-inflammatory, thereby promoting the obesity-linked disorders. In contrast, there are a small number of adipocytokines that exhibit antiinflammatory properties. It is now recognized that dysregulated production or secretion of adipocytokines caused by adipocyte dysfunction leads to the development of obesity-linked complications. In this review, we focus on the functional role of several adipocytokines in metabolic and cardiovascular diseases.

Indexed as

AdipokinesAdipose TissueAnimalsCardiovascular DiseasesHumansInflammationInflammation MediatorsMetabolic DiseasesObesityRisk FactorsAdipokinesInflammation Mediators

Identifiers

PMID22515108
PMCPMC4831247

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-ND
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.