Evidence map›Paper›PMID 22505686›Full record

ArticleJournal of bacteriology2012

SMU.152 acts as an immunity protein for mutacin IV.

Mohammad Shahnoor Hossain, Indranil Biswas

Open access · bronzeAbstract read
In one paragraph

Article in Journal of bacteriology, 2012. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
0.4field-weighted citation impact, top 35% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed, 26 citations in OpenAlex.

  1. Fluoride triggers lysis inProceedings of the National Academy of Sciences of the United States of America · 2025
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  3. Expanding the genetic toolbox for the obligate human pathogenFrontiers in bioengineering and biotechnology · 2024
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  4. Genomic and phenotypic characterization ofFrontiers in microbiology · 2022
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors at 1 institution in 1 country.

Mohammad Shahnoor HossainDepartment of Microbiology, Molecular Genetics and Immunology, University of Kansas Medical Center, Kansas City, Kansas, USA.
Indranil Biswas
University of Kansas Medical Center · US

Funding

Expression of clp genes in Streptococcus mutansR01DE021664 · NIDCR · UNIVERSITY OF KANSAS MEDICAL CENTER · PI BISWAS, INDRANIL · 2011 to 2015
$1.8M
Mechanisms of Virulence Gene Regulation in Streptococcus mutansR01DE016686 · NIDCR · UNIVERSITY OF SOUTH DAKOTA · PI BISWAS, INDRANIL · 2007 to 2011
$1.2M
NIDCR NIH HHS DE016686NIDCR NIH HHS DE021664NIDCR NIH HHS R01 DE016686NIDCR NIH HHS R01 DE021664
6 · The paper itself

Abstract

Streptococcus mutans, a principal causative agent of dental caries, secretes antimicrobial peptides known as mutacins to suppress the growth of competing species to establish a successful colonization. S. mutans UA159, a sequenced strain, produces at least two major mutacins, mutacins IV and V. Mutacin IV is a two-peptide mutacin encoded by nlmAB genes, which are mapped just upstream of a putative immunity-encoding gene SMU.152. Here we explored the function of SMU.152 as an immunity protein. We observed that overexpression of SMU.152 in two sensitive host strains converted the strains to become immune to mutacin IV. To identify the residues that are important for immunity function, we sequentially deleted residues from the C-terminal region of SMU.152. We observed that deletion of as few as seven amino acids, all of which are highly charged (KRRSKNK), drastically reduced the immunity function of the protein. Furthermore, we identified two other putative immunity proteins, SMU.1909 and SMU.925, which lack the last four charged residues (SKNK) that are present in SMU.152 but contain the KRR residues. Synthetic addition of SKNK residues to either SMU.1909 or SMU.925 to reconstitute the KRRSKNK motif and expressing these constructs in sensitive cells rendered the cells resistant to mutacin IV. We also demonstrated that deletion of Man-PTS system from a sensitive strain made the cells partially resistant to mutacin IV, indicating that the Man-PTS system plays a role in mutacin IV recognition.

Indexed as

Drug Resistance, BacterialAmino Acid SequenceAnti-Bacterial AgentsBacterial ProteinsBacteriocinsBase SequenceDental CariesHumansModels, MolecularMolecular Sequence DataSequence DeletionStreptococcus gordoniiStreptococcus mutansAnti-Bacterial AgentsBacterial ProteinsBacteriocinsmutacin IV

Identifiers

PMID22505686
PMCPMC3434736
OpenAlexW2058408817

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.