Evidence map›Paper›PMID 22493426›Full record

ArticleThe Journal of biological chemistry2012

IQGAP proteins reveal an atypical phosphoinositide (aPI) binding domain with a pseudo C2 domain fold.

Miles J Dixon, Alexander Gray, Martijn Schenning, Mark Agacan, Wolfram Tempel, Yufeng Tong, Lyudmila Nedyalkova, Hee-Won Park, Nicholas R Leslie, Daan M F van Aalten and 2 more

Open access · hybridAbstract read
In one paragraph

Article in The Journal of biological chemistry, 2012. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 17 papers.

0numbers the graph read from it
0cells of the map it votes in
17citing papers in PubMed
2.3field-weighted citation impact, top 13% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

17 citing papers in PubMed, 26 citations in OpenAlex.

  1. Queue Gaps Among the IQGAPs inInternational journal of molecular sciences · 2026
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  6. IQGAP1 is a phosphoinositide effector and kinase scaffold.Advances in biological regulation · 2016
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

12 authors at 4 institutions in 3 countries.

Miles J DixonDivision of Cell Signalling and Immunology, College of Life Sciences, University of Dundee, Dow St., Dundee DD1 5EH, Scotland, United Kingdom.
Alexander Gray
Martijn Schenning
Mark Agacan
Wolfram Tempel
Yufeng Tong
Lyudmila Nedyalkova
Hee-Won Park
Nicholas R Leslie
Daan M F van Aalten
C Peter Downes
Ian H Batty
Toxicologie, Pharmacologie et Signalisation Cellulaire · FRUniversity of Dundee · GBStructural Genomics Consortium · CAUniversity of Toronto · CA

Funding

CIHRMedical Research Council G0801865Medical Research Council G0900138Medical Research Council G9403619NCI NIH HHS Y1-CO-1020NIGMS NIH HHS Y1-GM-1104Wellcome Trust 083481Wellcome Trust 087590Wellcome Trust WT087590MA
6 · The paper itself

Abstract

Class I phosphoinositide (PI) 3-kinases act through effector proteins whose 3-PI selectivity is mediated by a limited repertoire of structurally defined, lipid recognition domains. We describe here the lipid preferences and crystal structure of a new class of PI binding modules exemplified by select IQGAPs (IQ motif containing GTPase-activating proteins) known to coordinate cellular signaling events and cytoskeletal dynamics. This module is defined by a C-terminal 105-107 amino acid region of which IQGAP1 and -2, but not IQGAP3, binds preferentially to phosphatidylinositol 3,4,5-trisphosphate (PtdInsP(3)). The binding affinity for PtdInsP(3), together with other, secondary target-recognition characteristics, are comparable with those of the pleckstrin homology domain of cytohesin-3 (general receptor for phosphoinositides 1), an established PtdInsP(3) effector protein. Importantly, the IQGAP1 C-terminal domain and the cytohesin-3 pleckstrin homology domain, each tagged with enhanced green fluorescent protein, were both re-localized from the cytosol to the cell periphery following the activation of PI 3-kinase in Swiss 3T3 fibroblasts, consistent with their common, selective recognition of endogenous 3-PI(s). The crystal structure of the C-terminal IQGAP2 PI binding module reveals unexpected topological similarity to an integral fold of C2 domains, including a putative basic binding pocket. We propose that this module integrates select IQGAP proteins with PI 3-kinase signaling and constitutes a novel, atypical phosphoinositide binding domain that may represent the first of a larger group, each perhaps structurally unique but collectively dissimilar from the known PI recognition modules.

Indexed as

3T3 CellsAmino Acid SequenceAnimalsBinding SitesCrystallographyGTPase-Activating ProteinsMiceMolecular Sequence DataPhosphatidylinositol 3-KinasesPhosphatidylinositolsProtein BindingProtein Structure, SecondaryProtein Structure, Tertiaryras GTPase-Activating ProteinsSignal TransductionGTPase-Activating ProteinsIqgap2 protein, mouseIQGAP3 protein, mouseIQ motif containing GTPase activating protein 1Phosphatidylinositol 3-KinasesPhosphatidylinositolsras GTPase-Activating Proteins

Identifiers

PMID22493426
PMCPMC3391087
OpenAlexW2101017386

What OpenQuestion holds

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LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.