Evidence map›Paper›PMID 22152698›Full record

ReviewZhongguo fei ai za zhi = Chinese journal of lung cancer2011

[Advances of DNA damage repair and Cisplatin resistance mechanisms in lung cancer].

Chunlan Tang, Heping Yang, Xiangdong Zhou

Open access · greenAbstract readEnglish AbstractReview
In one paragraph

Review in Zhongguo fei ai za zhi = Chinese journal of lung cancer, 2011. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers.

0numbers the graph read from it
0cells of the map it votes in
8citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

8 citing papers in PubMed, 10 citations in OpenAlex.

  1. Article
  2. Article
  3. Review
  4. Cancers · 2022
    Article
  5. Review
  6. Pharmacological Effects of Verticine: Current Status.Evidence-based complementary and alternative medicine : eCAM · 2019
    Review
  7. [ole of AGGF1 in DNA damage repair and modulating chemotherapy resistance in human colon cancer cellsNan fang yi ke da xue xue bao = Journal of Southern Medical University · 2018
    Article
  8. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 1 institution in 1 country.

Chunlan TangDepartment of Respiratory Medicine, Southwest Hospital, Third Military Medical University, Chongqing 400038, China.
Heping Yang
Xiangdong Zhou
Army Medical University · CN

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Lung cancer is the most common cause of death from cancer worldwide per year. Platinum-based combination chemotherapy is a main treatment of lung cancer. Cisplatin is adopted widely and used effectively in the first-line chemotherapy. Unfortunately, development of cisplatin resistance is a major obstacle to the success of lung caner. Cisplatin is a cell-cycle-non-specific cytotoxic drugs and its main target is DNA. Thus, defective DNA damage repair is one of the main mechanisms of cisplatin resistance. In this review, we will focus on the defective DNA damage repair in cisplatin resistance of lung cancer including nucleotide excision repair, DNA mismatch repair, DNA double-strand break repair and translesion synthesis.

Indexed as

DNA DamageDrug Resistance, NeoplasmAnimalsAntineoplastic AgentsCisplatinDNA RepairHumansLung NeoplasmsAntineoplastic AgentsCisplatin

Identifiers

PMID22152698
PMCPMC6000195
OpenAlexW281821295

What OpenQuestion holds

Textmetadata
LicenceCC BY
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.