ArticleCognitive neurodynamics2010
The modeling and simulation of visuospatial working memory.
Article in Cognitive neurodynamics, 2010. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
4 citing papers in PubMed.
- A Comparative Study of the Impact of Theta-Burst and High-Frequency Stimulation on Memory Performance.Frontiers in human neuroscience · 2016Article
- Perception of successive brief objects as a function of stimulus onset asynchrony: model experiments based on two-stage synchronization of neuronal oscillators.Cognitive neurodynamics · 2013Article
- Extended difference-of-Gaussians model incorporating cortical feedback for relay cells in the lateral geniculate nucleus of cat.Cognitive neurodynamics · 2012Article
- A minimal mechanistic model for temporal signal processing in the lateral geniculate nucleus.Cognitive neurodynamics · 2012Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
3 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Camperi and Wang (Comput Neurosci 5:383-405, 1998) presented a network model for working memory that combines intrinsic cellular bistability with the recurrent network architecture of the neocortex. While Fall and Rinzel (Comput Neurosci 20:97-107, 2006) replaced this intrinsic bistability with a biological mechanism-Ca(2+) release subsystem. In this study, we aim to further expand the above work. We integrate the traditional firing-rate network with Ca(2+) subsystem-induced bistability, amend the synaptic weights and suggest that Ca(2+) concentration only increase the efficacy of synaptic input but has nothing to do with the external input for the transient cue. We found that our network model maintained the persistent activity in response to a brief transient stimulus like that of the previous two models and the working memory performance was resistant to noise and distraction stimulus if Ca(2+) subsystem was tuned to be bistable.
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.