Evidence map›Paper›PMID 22120051›Full record

ArticleNature genetics2011

Fine points in mapping autoimmunity.

Constantin Polychronakos

Abstract readComment
PubMed Publisher
In one paragraph

Article in Nature genetics, 2011. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 12 papers.

0numbers the graph read from it
0cells of the map it votes in
12citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

12 citing papers in PubMed.

  1. Review
  2. Article
  3. Article
  4. Review
  5. Genetics of systemic sclerosis.Journal of scleroderma and related disorders · 2020
    Review
  6. Article
  7. Article
  8. Pathogenesis of Systemic Sclerosis.Frontiers in immunology · 2015
    Review
  9. Article
  10. Article
  11. Article
  12. Article
4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

1 author.

Constantin PolychronakosEndocrine Genetics Laboratory, Research Institute of the McGill University Health Centre, The Montreal Children’s Hospital, Québec, Canada. constantin.polychronakos@mcgill.ca

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

An efficient way to design genotyping arrays for fine mapping is to group phenotypes with common biology. The first application of the Immunochip to celiac disease provides an insightful view of what this strategy can achieve.

Indexed as

Polymorphism, Single NucleotideCeliac DiseaseHumans

Identifiers

PMID22120051

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.