Evidence map›Paper›PMID 22016586›Full record

ReviewThe Korean journal of internal medicine2011

Impact of air pollution on allergic diseases.

Hajime Takizawa

Abstract readReview
In one paragraph

Review in The Korean journal of internal medicine, 2011. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 30 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
30citing papers in PubMed, 1 pooled it
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

30 citing papers in PubMed, 1 synthesis or guideline pooled it.

  1. Pooled it
  2. Article
  3. Review
  4. Article
  5. Circulating Exosomes and Ambient Air Pollution Exposure in COPD.Chronic obstructive pulmonary diseases (Miami, Fla.) · 2023
    Article
  6. Article
  7. Review
  8. Article
  9. Sulfur dioxide and exacerbation of allergic respiratory diseases: A time-stratified case-crossover study.Journal of research in medical sciences : the official journal of Isfahan University of Medical Sciences · 2021
    Article
  10. Article
  11. Article
  12. Article
  13. Article
  14. Do helminth infections underpin urban-rural differences in risk factors for allergy-related outcomes?Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology · 2019
    Article
  15. Article
  16. Article
  17. Article
  18. Low Concentration PMClinical and experimental otorhinolaryngology · 2017
    Article
  19. Mitochondrial Function in Allergic Disease.Current allergy and asthma reports · 2017
    Review
  20. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

1 author.

Hajime TakizawaDepartment of Respiratory Medicine, Kyorin University School of Medicine, Tokyo, Japan. htakizawa-alg@umin.ac.jp

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The incidence of allergic diseases in most industrialized countries has increased. Although the exact mechanisms behind this rapid increase in prevalence remain uncertain, a variety of air pollutants have been attracting attention as one causative factor. Epidemiological and toxicological research suggests a causative relationship between air pollution and the increased incidence of asthma, allergic rhinitis, and other allergic disorders. These include ozone, nitrogen dioxide and, especially particulate matter, produced by traffic-related and industrial activities. Strong epidemiological evidence supports a relationship between air pollution and the exacerbation of asthma and other respiratory diseases. Recent studies have suggested that air pollutants play a role in the development of asthma and allergies. Researchers have elucidated the mechanisms whereby these pollutants induce adverse effects; they appear to affect the balance between antioxidant pathways and airway inflammation. Gene polymorphisms involved in antioxidant pathways can modify responses to air pollution exposure. While the characterization and monitoring of pollutant components currently dictates pollution control policies, it will be necessary to identify susceptible subpopulations to target therapy/prevention of pollution-induced respiratory diseases.

Indexed as

Environmental ExposureAir PollutantsAnimalsGene-Environment InteractionHumansHypersensitivityOxidative StressRisk AssessmentRisk FactorsAir PollutantsAir pollutantsAntioxidantAsthmaParticulate matterVehicle emissions

Identifiers

PMID22016586
PMCPMC3192198

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.