ArticleMathematical biosciences2011
Extinction models for cancer stem cell therapy.
Article in Mathematical biosciences, 2011. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 12 papers.
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Who cites it
12 citing papers in PubMed.
- The Evolution and Ecology of Resistance in Cancer Therapy.Cold Spring Harbor perspectives in medicine · 2020Review
- Duration of a minor epidemic.Infectious Disease Modelling · 2018Article
- Modeling of Interactions between Cancer Stem Cells and their Microenvironment: Predicting Clinical Response.Methods in molecular biology (Clifton, N.J.) · 2018Review
- Sampling from single-cell observations to predict tumor cell growthOncotarget · 2017Article
- Modeling head and neck cancer stem cell-mediated tumorigenesis.Cellular and molecular life sciences : CMLS · 2016Review
- Curcumin Enhanced Busulfan-Induced Apoptosis through Downregulating the Expression of Survivin in Leukemia Stem-Like KG1a Cells.BioMed research international · 2015Article
- Effect of dedifferentiation on time to mutation acquisition in stem cell-driven cancers.PLoS computational biology · 2014Article
- Cell surface markers of cancer stem cells: diagnostic macromolecules and targets for drug delivery.Drug delivery and translational research · 2013Article
- Systems and Network Pharmacology Approaches to Cancer Stem Cells Research and Therapy.Journal of stem cell research & therapy · 2012Article
- Article
- Differential destruction of stem cells: implications for targeted cancer stem cell therapy.Cancer research · 2009Article
- Fitting Birth-Death Processes to Panel Data with Applications to Bacterial DNA Fingerprinting.The annals of applied statisticsArticle
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4 authors.
Funding
Abstract
Cells with stem cell-like properties are now viewed as initiating and sustaining many cancers. This suggests that cancer can be cured by driving these cancer stem cells to extinction. The problem with this strategy is that ordinary stem cells are apt to be killed in the process. This paper sets bounds on the killing differential (difference between death rates of cancer stem cells and normal stem cells) that must exist for the survival of an adequate number of normal stem cells. Our main tools are birth-death Markov chains in continuous time. In this framework, we investigate the extinction times of cancer stem cells and normal stem cells. Application of extreme value theory from mathematical statistics yields an accurate asymptotic distribution and corresponding moments for both extinction times. We compare these distributions for the two cell populations as a function of the killing rates. Perhaps a more telling comparison involves the number of normal stem cells NH at the extinction time of the cancer stem cells. Conditioning on the asymptotic time to extinction of the cancer stem cells allows us to calculate the asymptotic mean and variance of NH. The full distribution of NH can be retrieved by the finite Fourier transform and, in some parameter regimes, by an eigenfunction expansion. Finally, we discuss the impact of quiescence (the resting state) on stem cell dynamics. Quiescence can act as a sanctuary for cancer stem cells and imperils the proposed therapy. We approach the complication of quiescence via multitype branching process models and stochastic simulation. Improvements to the τ-leaping method of stochastic simulation make it a versatile tool in this context. We conclude that the proposed therapy must target quiescent cancer stem cells as well as actively dividing cancer stem cells. The current cancer models demonstrate the virtue of attacking the same quantitative questions from a variety of modeling, mathematical, and computational perspectives.
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