ReviewImmunology and allergy clinics of North America2011
Pathogenesis and disease mechanisms of occupational asthma.
Review in Immunology and allergy clinics of North America, 2011. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 23 papers.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
23 citing papers in PubMed, 50 citations in OpenAlex.
- Using Multiple Diagnostic Methods for Occupational Asthma Assessment.The clinical respiratory journal · 2026Article
- Clinical characteristics and risk factors in pneumoconiosis patients with asthma.Scientific reports · 2025Article
- Bioaerosol Exposures and Respiratory Diseases in Cannabis Workers.Current allergy and asthma reports · 2024Review
- Maternal occupational exposure to asthmogenic during pregnancy and the future risk of asthma in children: A meta-analysis.Turkish journal of obstetrics and gynecology · 2024Article
- Mechanistic and Therapeutic Approaches to Occupational Exposure-Associated Allergic and Non-Allergic Asthmatic Disease.Current allergy and asthma reports · 2023Review
- Early IL-17A Prevention Rather Than Late IL-17A Neutralization Attenuates Toluene Diisocyanate-Induced Mixed Granulocytic Asthma.Allergy, asthma & immunology research · 2022Article
- Association ofCanadian respiratory journal · 2022Article
- Associations Between Glutathione-S-Transferase Genotypes and Bronchial Hyperreactivity Patients With Di-isocyanate Induced Asthma. A Follow-Up Study.Frontiers in medicine · 2019Article
- Genetic variants with gene regulatory effects are associated with diisocyanate-induced asthma.The Journal of allergy and clinical immunology · 2018Article
- Role of clusterin/progranulin in toluene diisocyanate-induced occupational asthma.Experimental & molecular medicine · 2018Article
- Polymerization of hexamethylene diisocyanate in solution and a 260.23 m/z [M+H]Analytical biochemistry · 2018Article
- Diabetes Downregulates Allergen-Induced Airway Inflammation in Mice.Mediators of inflammation · 2018Article
- Polycyclic aromatic hydrocarbons exposure, oxidative stress, and asthma in children.International archives of occupational and environmental health · 2017Article
- Oxidative Stress-Related Genetic Variants May Modify Associations of Phthalate Exposures with Asthma.International journal of environmental research and public health · 2017Article
- The Role and Immunobiology of Eosinophils in the Respiratory System: a Comprehensive Review.Clinical reviews in allergy & immunology · 2016Review
- N-Acetyltransferase 2 Genotypes Are Associated With Diisocyanate-Induced Asthma.Journal of occupational and environmental medicine · 2015Article
- Phenotypes, Risk Factors, and Mechanisms of Adult-Onset Asthma.Mediators of inflammation · 2015Review
- Occupational asthma: an overview.Current allergy and asthma reports · 2014Review
- Genetic variants in the major histocompatibility complex class I and class II genes are associated with diisocyanate-induced Asthma.Journal of occupational and environmental medicine · 2014Article
- Experiences of racism and the incidence of adult-onset asthma in the Black Women's Health Study.Chest · 2014Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
3 authors at 2 institutions in 1 country.
Funding
Abstract
Occupational asthma (OA) is one of the most common forms of work-related lung disease in all industrialized nations. The clinical management of patients with OA depends on an understanding of the multifactorial pathogenetic mechanisms that can contribute to this disease. This article discusses the various immunologic and nonimmunologic mechanisms and genetic susceptibility factors that drive the inflammatory processes of OA.
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.