Evidence map›Paper›PMID 21965670›Full record

ArticleThe Journal of biological chemistry2011

The C-terminal αI domain linker as a critical structural element in the conformational activation of αI integrins.

Gabriele Weitz-Schmidt, Thomas Schürpf, Timothy A Springer

Open access · hybridAbstract read
In one paragraph

Article in The Journal of biological chemistry, 2011. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers.

0numbers the graph read from it
0cells of the map it votes in
8citing papers in PubMed
0.5field-weighted citation impact, top 39% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

8 citing papers in PubMed, 12 citations in OpenAlex.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 2 institutions in 1 country.

Gabriele Weitz-SchmidtImmune Disease Institute, Children's Hospital Boston and Department of Pathology, Harvard Medical School, Boston Massachusetts 02115; University Basel, PharmaCenter, Klingelbergstr. 50-70, 4056 Basel, Switzerland. Electronic address: gabriele.weitz-schmidt@unibas.ch.
Thomas SchürpfImmune Disease Institute, Children's Hospital Boston and Department of Pathology, Harvard Medical School, Boston Massachusetts 02115.
Timothy A SpringerImmune Disease Institute, Children's Hospital Boston and Department of Pathology, Harvard Medical School, Boston Massachusetts 02115. Electronic address: springer@idi.harvard.edu.
Boston Children's Hospital · USHarvard University · US

Funding

LYMPHOCYTE FUNCTION ASSOCIATED ANTIGENSR37CA031798 · NCI · IMMUNE DISEASE INSTITUTE, INC. · PI SPRINGER, TIMOTHY A · 2004 to 2013
$8.3M
LYMPHOCYTE FUNCTION-ASSOCIATED ANTIGENSR01CA031798 · NCI · IMMUNE DISEASE INSTITUTE, INC. · PI SPRINGER, TIMOTHY A · 1985 to 2018
$5.1M
NCI NIH HHS CA31798NCI NIH HHS R01 CA031798NCI NIH HHS R37 CA031798
6 · The paper itself

Abstract

The activation of α/β heterodimeric integrins is the result of highly coordinated rearrangements within both subunits. The molecular interactions between the two subunits, however, remain to be characterized. In this study, we use the integrin α(L)β(2) to investigate the functional role of the C-linker polypeptide that connects the C-terminal end of the inserted (I) domain with the β-propeller domain on the α subunit and is located at the interface with the βI domain of the β chain. We demonstrate that shortening of the C-linker by eight or more amino acids results in constitutively active α(L)β(2) in which the αI domain is no longer responsive to the regulation by the βI domain. Despite this intersubunit uncoupling, both I domains remain individually sensitive to intrasubunit conformational changes induced by allosteric modulators. Interestingly, the length and not the sequence of the C-linker appears to be critical for its functionality in α/β intersubunit communication. Using two monoclonal antibodies (R7.1 and CBR LFA-1/1) we further demonstrate that shortening of the C-linker results in the gradual loss of combinational epitopes that require both the αI and β-propeller domains for full reactivity. Taken together, our findings highlight the role of the C-linker as a spring-like element that allows relaxation of the αI domain in the resting state and controlled tension of the αI domain during activation, exerted by the β chain.

Indexed as

Allosteric SiteAnimalsAntibodies, MonoclonalCell AdhesionCell LineCell MembraneDimerizationHumansIntegrin alpha1Lymphocyte Function-Associated Antigen-1MiceMicroscopy, FluorescenceProtein BindingProtein ConformationProtein Structure, TertiaryTransfectionAntibodies, MonoclonalIntegrin alpha1Lymphocyte Function-Associated Antigen-1

Identifiers

PMID21965670
PMCPMC3234956
OpenAlexW2103256953

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.