Evidence map›Paper›PMID 21911966›Full record

ReviewThe Indian journal of medical research2011

An understanding of the genetic basis of asthma.

Mahdi Bijanzadeh, Padukudru A Mahesh, Nallur B Ramachandra

Abstract readReview
In one paragraph

Review in The Indian journal of medical research, 2011. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 34 papers.

0numbers the graph read from it
0cells of the map it votes in
34citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

34 citing papers in PubMed.

  1. Article
  2. A Functional Variant ofJournal of asthma and allergy · 2026
    Article
  3. Article
  4. Review
  5. Review
  6. Article
  7. Review
  8. Association betweenDiagnostics (Basel, Switzerland) · 2023
    Article
  9. Article
  10. Article
  11. Review
  12. Article
  13. Review
  14. Article
  15. The Association of IgE Levels withJournal of personalized medicine · 2021
    Article
  16. Review
  17. Article
  18. Haplotype analysis ofThe Indian journal of medical research · 2019
    Article
  19. Article
  20. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors.

Mahdi BijanzadehDepartment of Studies in Zoology, University of Mysore, Mysore, India.
Padukudru A Mahesh
Nallur B Ramachandra

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Asthma is the most common chronic childhood disease in developed nations and its prevalence has increased in the world over the last 25 years. It is a complex disease with both genetic and environmental risk factors. Asthma is caused by multiple interacting genes, some having a protective effect and others contributing to the disease pathogenesis, with each gene having its own tendency to be influenced by the environment. This article reviews the current state of the genetics of asthma in six categories, viz. epidemiology, management, aetiology, family and twin studies, segregation and linkage studies, and candidate genes and single nucleotide polymorphisms (SNPs).

Indexed as

AsthmaCausalityDiseases in TwinsGene-Environment InteractionGenetic LinkageGenetic Predisposition to DiseaseHumansPolymorphism, Single NucleotidePrevalence

Identifiers

PMID21911966
PMCPMC3181014

What OpenQuestion holds

Textmetadata
LicenceCC BY-NC-SA
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.