ArticleMolecular biology of the cell2011
The chromatin remodeler ISWI regulates the cellular response to hypoxia: role of FIH.
Article in Molecular biology of the cell, 2011. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 22 papers.
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Who cites it
22 citing papers in PubMed, 38 citations in OpenAlex.
- KDM2B controls HIF levels and activity through its JmjC and CxxC domains.The Biochemical journal · 2026Article
- Regulation of chromatin accessibility by hypoxia and HIF.The Biochemical journal · 2022Article
- Epigenetic Regulation During Hypoxia and Its Implications in Cancer.Sub-cellular biochemistry · 2022Article
- PERK/eIF2α signaling inhibits HIF-induced gene expression during the unfolded protein response via YB1-dependent regulation of HIF1α translation.Nucleic acids research · 2018Article
- Hypoxia and Chromatin: A Focus on Transcriptional Repression Mechanisms.Biomedicines · 2018Review
- Epigenetic regulators: multifunctional proteins modulating hypoxia-inducible factor-α protein stability and activity.Cellular and molecular life sciences : CMLS · 2018Review
- The epigenetic landscape related to reactive oxygen species formation in the cardiovascular system.British journal of pharmacology · 2017Review
- Potent and selective chemical probe of hypoxic signalling downstream of HIF-α hydroxylation via VHL inhibition.Nature communications · 2016Article
- CDK-dependent phosphorylation of PHD1 on serine 130 alters its substrate preference in cells.Journal of cell science · 2016Article
- Dose-dependent effects of allopurinol on human foreskin fibroblast cells and human umbilical vein endothelial cells under hypoxia.PloS one · 2015Article
- Developmental programming: the role of growth hormone.Journal of animal science and biotechnology · 2015Article
- Cezanne (OTUD7B) regulates HIF-1α homeostasis in a proteasome-independent manner.EMBO reports · 2014Article
- Chromatin and oxygen sensing in the context of JmjC histone demethylases.The Biochemical journal · 2014Review
- Cell cycle progression in response to oxygen levels.Cellular and molecular life sciences : CMLS · 2014Review
- SiRNA screening to identify ubiquitin and ubiquitin-like system regulators of biological pathways in cultured mammalian cells.Journal of visualized experiments : JoVE · 2014Article
- Regulation of CDKN2B expression by interaction of Arnt with Miz-1--a basis for functional integration between the HIF and Myc gene regulatory pathways.Molecular cancer · 2014Article
- The roles of Jumonji-type oxygenases in human disease.Epigenomics · 2014Review
- PITX1, a specificity determinant in the HIF-1α-mediated transcriptional response to hypoxia.Cell cycle (Georgetown, Tex.) · 2014Article
- IFNβ autocrine feedback is required to sustain TLR induced production of MCP-1 in macrophages.FEBS letters · 2013Article
- The P-body component USP52/PAN2 is a novel regulator of HIF1A mRNA stability.The Biochemical journal · 2013Article
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Authors and funding
3 authors at 1 institution in 1 country.
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Abstract
The hypoxia-inducible factor (HIF) is a master regulator of the cellular response to hypoxia. Its levels and activity are controlled by dioxygenases called prolyl-hydroxylases and factor inhibiting HIF (FIH). To activate genes, HIF has to access sequences in DNA that are integrated in chromatin. It is known that the chromatin-remodeling complex switch/sucrose nonfermentable (SWI/SNF) is essential for HIF activity. However, no additional information exists about the role of other chromatin-remodeling enzymes in hypoxia. Here we describe the role of imitation switch (ISWI) in the cellular response to hypoxia. We find that unlike SWI/SNF, ISWI depletion enhances HIF activity without altering its levels. Furthermore, ISWI knockdown only alters a subset of HIF target genes. Mechanistically, we find that ISWI is required for full expression of FIH mRNA and protein levels by changing RNA polymerase II loading to the FIH promoter. Of interest, exogenous FIH can rescue the ISWI-mediated upregulation of CA9 but not BNIP3, suggesting that FIH-independent mechanisms are also involved. Of importance, ISWI depletion alters the cellular response to hypoxia by reducing autophagy and increasing apoptosis. These results demonstrate a novel role for ISWI as a survival factor during the cellular response to hypoxia.
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