Evidence map›Paper›PMID 21814462›Full record

ReviewInternational journal of chronic obstructive pulmonary disease2011

Improved patient outcome with smoking cessation: when is it too late?

Jane Wu, Don D Sin

Abstract readReview
In one paragraph

Review in International journal of chronic obstructive pulmonary disease, 2011. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 34 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
34citing papers in PubMed, 1 pooled it
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

34 citing papers in PubMed, 1 synthesis or guideline pooled it.

  1. Pooled it
  2. Trial
  3. Article
  4. Article
  5. Article
  6. Mendelian causes of early-onset emphysema: a review of the current literature.European respiratory review : an official journal of the European Respiratory Society · 2025
    Review
  7. Article
  8. Article
  9. Animal models and mechanisms of tobacco smoke-induced chronic obstructive pulmonary disease (COPD).Journal of toxicology and environmental health. Part B, Critical reviews · 2023
    Review
  10. Article
  11. Risk factor control among heart failure patients in the United States: Results from the NHANES 1999-2018.International journal of cardiology. Cardiovascular risk and prevention · 2022
    Article
  12. Impact of Smoking Cessation on Periodontal Tissues.International dental journal · 2022
    Review
  13. Article
  14. Article
  15. Article
  16. Article
  17. Article
  18. The cardiovascular burden of light smoking.Archives of medical sciences. Atherosclerotic diseases · 2021
    Article
  19. Article
  20. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Jane WuDepartment of Medicine (Division of Respirology), The University of British Columbia, Vancouver, BC, Canada.
Don D Sin

Funding

Canadian Institutes of Health Research
6 · The paper itself

Abstract

Smoking is the leading modifiable risk factor for chronic obstructive pulmonary disease (COPD), cardiovascular disease (CVD), and lung cancer. Smoking cessation is the only proven way of modifying the natural course of COPD. It is also the most effective way of reducing the risk for myocardial infarction and lung cancer. However, the full benefits of tobacco treatment may not be realized until many years of abstinence. All patients with COPD, regardless of severity, appear to benefit from tobacco treatment. Similarly, patients with recent CVD events also benefit from tobacco treatment. The risk of total mortality and rate of recurrence of lung cancer is substantially lower in smokers who manage to quit smoking following the diagnosis of early stage lung cancer or small cell lung cancer. Together, these data suggest that tobacco treatment is effective both as a primary and a secondary intervention in reducing total morbidity and mortality related to COPD, CVD, and lung cancer. In this paper, we summarize the evidence for tobacco treatment and the methods by which smoking cessation can be promoted in smokers with lung disease.

Indexed as

Smoking CessationHealth PromotionHumansLung NeoplasmsMyocardial InfarctionPulmonary Disease, Chronic ObstructiveRisk AssessmentRisk FactorsRisk Reduction BehaviorSmokingTime FactorsTobacco Use DisorderTreatment OutcomeCOPDlung cancersmoking cessationtobacco treatment

Identifiers

PMID21814462
PMCPMC3144846

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.