Evidence map›Paper›PMID 21671475›Full record

ArticleInternational journal of cancer2012

An American founder mutation in MLH1.

Jerneja Tomsic, Sandya Liyanarachchi, Heather Hampel, Monika Morak, Brittany C Thomas, Victoria M Raymond, Anu Chittenden, Hans K Schackert, Stephen B Gruber, Sapna Syngal and 4 more

Open access · bronzeAbstract read
In one paragraph

Article in International journal of cancer, 2012. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 10 papers.

0numbers the graph read from it
0cells of the map it votes in
10citing papers in PubMed
0.8field-weighted citation impact, top 27% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

10 citing papers in PubMed, 18 citations in OpenAlex.

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  5. A Novel GermlineGenes · 2020
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

14 authors at 9 institutions in 3 countries.

Jerneja TomsicHuman Cancer Genetics Program, Comprehensive Cancer Center, The Ohio State University, Columbus, OH 43210, USA.
Sandya Liyanarachchi
Heather Hampel
Monika Morak
Brittany C Thomas
Victoria M Raymond
Anu Chittenden
Hans K Schackert
Stephen B Gruber
Sapna Syngal
Alessandra Viel
Elke Holinski-Feder
Stephen N Thibodeau
Albert de la Chapelle
Cancer Genetics (United States) · USMayo Clinic · USMedical Genetics Center · DEThe Ohio State University · USUniversity of Michigan–Ann Arbor · USDana-Farber Cancer Institute · USHarvard University · USIstituti di Ricovero e Cura a Carattere Scientifico · ITTU Dresden · DE

Funding

Translational Therapeutics Research Program (TT)P30CA016058 · NCI · OHIO STATE UNIVERSITY · PI Tasleem J Padamsee · 1985 to 2026
$132.3M
INCIDENCE AND MOLECULAR SCREENING FOR HEREDITARY CANCERR01CA067941 · NCI · OHIO STATE UNIVERSITY · PI DE LA CHAPELLE, ALBERT · 2000 to 2004
$2.9M
NATURAL HISTORY AND INCIDENCE OF HEREDITARY COLON CANCERU01CA067941 · NCI · UNIVERSITY OF HELSINKI · PI DE LA CHAPELLE, ALBERT · 1996 to 1999
–
NCI NIH HHS CA16058NCI NIH HHS CA67941NCI NIH HHS P30 CA016058NCI NIH HHS R01 CA067941NCI NIH HHS U01 CA067941
6 · The paper itself

Abstract

Mutations in the mismatch repair genes cause Lynch syndrome (LS), conferring high risk of colorectal, endometrial and some other cancers. After the same splice site mutation in the MLH1 gene (c.589-2A>G) had been observed in four ostensibly unrelated American families with typical LS cancers, its occurrence in comprehensive series of LS cases (Mayo Clinic, Germany and Italy) was determined. It occurred in 10 out of 995 LS mutation carriers (1.0%) diagnosed in the Mayo Clinic diagnostic laboratory. It did not occur among 1,803 cases tested for MLH1 mutations by the German HNPCC consortium, while it occurred in three probands and an additional five family members diagnosed in Italy. In the U.S., the splice site mutation occurs on a large (∼4.8 Mb) shared haplotype that also harbors the variant c.2146G>A, which predicts a missense change in codon 716 referred to here as V716M. In Italy, it occurs on a different, shorter shared haplotype (∼2.2 Mb) that does not carry V716M. The V716M variant was found to be present by itself in the U.S., German and Italian populations with individuals sharing a common haplotype of 280 kb, allowing us to calculate that the variant arose around 5,600 years ago (225 generations; 95% confidence interval 183-272). The splice site mutation in America arose or was introduced some 450 years ago (18 generations; 95% confidence interval 14-23); it accounts for 1.0% all LS in the Unites States and can be readily screened for.

Indexed as

MutationAdaptor Proteins, Signal TransducingAdultAgedAllelesColorectal Neoplasms, Hereditary NonpolyposisExonsFemaleGenetic Predisposition to DiseaseGermanyHaplotypesHeterozygoteHumansIntronsItalyMaleAdaptor Proteins, Signal TransducingMLH1 protein, humanMutL Protein Homolog 1Nuclear ProteinsRNA Splice Sites

Identifiers

PMID21671475
PMCPMC3266960
OpenAlexW2073330380

What OpenQuestion holds

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Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.