Evidence map›Paper›PMID 21621699›Full record

ReviewThe Surgical clinics of North America2011

What, how, and how much should patients with burns be fed?

Felicia N Williams, Ludwik K Branski, Marc G Jeschke, David N Herndon

Open access · greenAbstract readReview
In one paragraph

Review in The Surgical clinics of North America, 2011. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 14 papers.

0numbers the graph read from it
0cells of the map it votes in
14citing papers in PubMed
2.4field-weighted citation impact, top 12% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

14 citing papers in PubMed, 71 citations in OpenAlex.

  1. Review
  2. Article
  3. Review
  4. Review
  5. Article
  6. Review
  7. [Research advances on early enteral nutritional therapy in severe burn patients].Zhonghua shao shang za zhi = Zhonghua shaoshang zazhi = Chinese journal of burns · 2021
    Article
  8. Article
  9. Article
  10. Article
  11. Early Enteral Nutrition for Burn Injury.Advances in wound care · 2014
    Review
  12. Review
  13. Burns: Pathophysiology of Systemic Complications and Current Management.Journal of burn care & research : official publication of the American Burn Association
    Review
  14. Postburn Hypermetabolism: Past, Present, and Future.Journal of burn care & research : official publication of the American Burn Association
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 2 institutions in 2 countries.

Felicia N WilliamsDepartment of Surgery, Shriners Hospital for Children and University of Texas Medical Branch, 815 Market Street, Galveston, TX 77550, USA.
Ludwik K Branski
Marc G Jeschke
David N Herndon
Shriners Hospitals for Children - Galveston · USSunnybrook Health Science Centre · CA

Funding

Inflammation and the Response to InjuryU54GM062119 · NIGMS · MASSACHUSETTS GENERAL HOSPITAL · PI DAVIS, RONALD WAYNE · 2001 to 2010
$78.6M
Project 9: The role of androgen/corticosteroid in fat, muscle and glucose metabolism of burn victimsP50GM060338 · NIGMS · UNIVERSITY OF TEXAS MEDICAL BR GALVESTON · PI HERNDON, DAVID N. · 2000 to 2017
$24.2M
MODULATION OF THE POSTBURN HYPERMETABOLIC RESPONSER01GM056687 · NIGMS · UNIVERSITY OF TEXAS MEDICAL BR GALVESTON · PI HERNDON, DAVID N. · 1998 to 2017
$9.0M
Oxandrolone and Exercise: A potent therapy in the rehabilitation from burnsR01HD049471 · NICHD · UNIVERSITY OF TEXAS MED BR GALVESTON · PI SUMAN-VEJAS, OSCAR E. · 2006 to 2019
$5.8M
POSTDOCTORAL TRAINING IN TRAUMA &BURNST32GM008256 · NIGMS · UNIVERSITY OF TEXAS MEDICAL BR GALVESTON · PI Celeste Campbell Finnerty, Oscar E. Suman-Vejas · 1990 to 2026
$4.0M
NICHD NIH HHS R01 HD049471NICHD NIH HHS R01-HD049471NIGMS NIH HHS 1P50GM60338-01NIGMS NIH HHS 2T32GM0825611NIGMS NIH HHS 5R01GM56687-03NIGMS NIH HHS P50 GM060338NIGMS NIH HHS R01 GM056687NIGMS NIH HHS R01-GM56687NIGMS NIH HHS T32 GM008256NIGMS NIH HHS U54 GM062119NIGMS NIH HHS U54/GM62119PHS HHS H133A020102PHS HHS H133A70019
6 · The paper itself

Abstract

The hypermetabolic response to severe burn injury is characterized by hyperdynamic circulation and profound metabolic, physiologic, catabolic, and immune system derangements. Failure to satisfy overwhelming energy and protein requirements after, and during, severe burn injury results in multiorgan dysfunction, increased susceptibility to infection, and death. Attenuation of the hypermetabolic response by various pharmacologic modalities is emerging as an essential component of the management of patients with severe burn injury. This review focuses on the more recent advances in therapeutic strategies to attenuate the hypermetabolic response and its postburn-associated insulin resistance.

Indexed as

Nutritional SupportAnimalsBlood GlucoseBurnsCatecholaminesEnteral NutritionHumansHypoglycemic AgentsInsulin ResistanceLipolysisLiverMetforminMultiple Organ FailureMuscle, SkeletalParenteral Nutrition, TotalWound HealingBlood GlucoseCatecholaminesHypoglycemic AgentsMetformin

Identifiers

PMID21621699
PMCPMC3255093
OpenAlexW2148445123

What OpenQuestion holds

Texttitle and abstract
LicenceTDM
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.