ArticleDermato-endocrinology2011
From the bench to emerging new clinical concepts: Our present understanding of the importance of the vitamin D endocrine system (VDES) for skin cancer.
Article in Dermato-endocrinology, 2011. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
1 citing paper in PubMed.
- 1α,25(OH)2-dihydroxyvitamin D3/VDR protects the skin from UVB-induced tumor formation by interacting with the β-catenin pathway.The Journal of steroid biochemistry and molecular biology · 2013Review
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
2 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
It is well accepted that growth and differentiation of keratinocytes and other cell types are regulated by 1,25-dihydroxyvitamin D(3) [1,25(OH)(2)D(3)], the active form of vitamin D. Moreover, growing evidence now indicates that the UV-B-mediated cutaneous photosynthesis of vitamin D [and the consecutive cutaneous production of 1,25(OH)(2)D(3))] represents an evolutionary highly-conserved endocrine system that protects the skin against environmental hazards that may promote skin cancerogenesis, including ultraviolet and ionizing radiation. It is the aim of this review to summarize our present understanding of the cutaneous vitamin D endocrine system (VDES) and its importance for the prevention of skin cancerogenesis.
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