ArticleKorean journal of pediatrics2011
Renal transplantation in a patient with Bartter syndrome and glomerulosclerosis.
Article in Korean journal of pediatrics, 2011. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 10 papers.
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Who cites it
10 citing papers in PubMed, 18 citations in OpenAlex.
- Complexities of Bartter Syndrome Type III: A Case Study in Jordan.Oxford medical case reports · 2025Article
- Type 1 Bartter syndrome presenting as primary diabetes insipidus: a rare Case Report with 8-year follow-up.Frontiers in genetics · 2025Article
- Successful kidney transplantation in a patient with late-onset type II Bartter syndrome: A rare case report.Clinical nephrology. Case studies · 2025Article
- Genetic Heterogeneity in Bartter Syndrome: Clinical and Practical Importance.Frontiers in pediatrics · 2022Review
- Bartter's syndrome: clinical findings, genetic causes and therapeutic approach.World journal of pediatrics : WJP · 2021Review
- Bartter and Gitelman syndromes: Questions of class.Pediatric nephrology (Berlin, Germany) · 2020Review
- Two novel mutations in theBMJ case reports · 2020Article
- Bartter syndrome: causes, diagnosis, and treatment.International journal of nephrology and renovascular disease · 2018Review
- Clinical and Genetic Spectrum of Bartter Syndrome Type 3.Journal of the American Society of Nephrology : JASN · 2017Article
- An Adult Case of Bartter Syndrome Type III Presenting with Proteinuria.Journal of pathology and translational medicine · 2016Article
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Authors and funding
9 authors at 3 institutions in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Bartter syndrome (BS) is a clinically and genetically heterogeneous inherited renal tube disorder characterized by renal salt wasting, hypokalemic metabolic alkalosis and normotensive hyperreninemic hyperaldosteronism. There have been several case reports of BS complicated by focal segmental glomerulosclerosis (FSGS). Here, we have reported the case of a BS patient who developed FSGS and subsequent end-stage renal disease (ESRD) and provided a brief literature review. The patient presented with classic BS at 3 months of age and developed proteinuria at 7 years. Renal biopsy performed at 11 years of age revealed a FSGS perihilar variant. Hemodialysis was initiated at 11 years of age, and kidney transplantation was performed at 16 years of age. The post-transplantation course has been uneventful for more than 3 years with complete disappearance of BS without the recurrence of FSGS. Genetic study revealed a homozygous p.Trp(TGG)610Stop(TGA) mutation in the CLCNKB gene. In summary, BS may be complicated by secondary FSGS due to the adaptive response to chronic salt-losing nephropathy, and FSGS may progress to ESRD in some patients. Renal transplantation in patients with BS and ESRD results in complete remission of BS.
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