ReviewCellular & molecular immunology2011
Dysfunctions of the Iga system: a common link between intestinal and renal diseases.
Review in Cellular & molecular immunology, 2011. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 19 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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Who cites it
19 citing papers in PubMed, 36 citations in OpenAlex.
- Nutritional needs of non-essential amino acids for intestinal health and growth performance of nursery pigs fed diets without soybean meal.Journal of animal science · 2026Article
- Post-weaning diarrhea caused by F18Microbiome research reports · 2025Review
- Inflammatory Bowel Diseases and Nephropathies: Exploring the Gut-Kidney Axis.Life (Basel, Switzerland) · 2024Review
- Causal association between inflammatory bowel disease and IgA nephropathy: A bidirectional two-sample Mendelian randomization study.Frontiers in genetics · 2022Article
- Intestinal Barrier in Human Health and Disease.International journal of environmental research and public health · 2021Review
- IgA glycosylation and immune complex formation in IgAN.Seminars in immunopathology · 2021Review
- Aberrantly Glycosylated IgA1 in IgA Nephropathy: What We Know and What We Don't Know.Journal of clinical medicine · 2021Review
- Celiac Disease-Type Tissue Transglutaminase Autoantibody Deposits in Kidney Biopsies of Patients with IgA Nephropathy.Nutrients · 2021Article
- Re-detectable positive SARS-CoV-2 RNA tests in patients who recovered from COVID-19 with intestinal infection.Protein & cell · 2021Article
- Simultaneous Immunoglobulin A and G Glycopeptide Profiling for High-Throughput Applications.Analytical chemistry · 2020Article
- Perspectives on how mucosal immune responses, infections and gut microbiome shape IgA nephropathy and future therapies.Theranostics · 2020Review
- Epigenetic Mechanisms and Posttranslational Modifications in Systemic Lupus Erythematosus.International journal of molecular sciences · 2019Review
- IgA and FcαRI: Pathological Roles and Therapeutic Opportunities.Frontiers in immunology · 2019Review
- Environmental Enrichment Induces Pericyte and IgA-Dependent Wound Repair and Lifespan Extension in a Colon Tumor Model.Cell reports · 2017Article
- Role of IgA receptors in the pathogenesis of IgA nephropathy.Journal of nephrology · 2016Review
- Gluten exacerbates IgA nephropathy in humanized mice through gliadin-CD89 interaction.Kidney international · 2015Article
- Salivary Immunoglobulin A Secretion Rate Is Negatively Associated with Cancer Mortality: The West of Scotland Twenty-07 Study.PloS one · 2015Article
- The kinase TBK1 controls IgA class switching by negatively regulating noncanonical NF-κB signaling.Nature immunology · 2012Article
- A novel differential diagnostic model based on multiple biological parameters for immunoglobulin A nephropathy.BMC medical informatics and decision making · 2012Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
3 authors at 2 institutions in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Immunoglobulin A (Iga)-isotype antibodies play an important role in immunity owing to their structure, glycosylation, localization and receptor interactions. Dysfunctions in this system can lead to multiple types of pathology. This review describes the characteristics of Iga and discusses the involvement of abnormalities in the Iga system on the development of celiac disease and Iga nephropathy.
Indexed as
Identifiers
What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.