Evidence map›Paper›PMID 21258997›Full record

ArticleTAG. Theoretical and applied genetics. Theoretische und angewandte Genetik2011

Quantitative trait loci analysis for resistance to Cephalosporium stripe, a vascular wilt disease of wheat.

Martin C Quincke, C James Peterson, Robert S Zemetra, Jennifer L Hansen, Jianli Chen, Oscar Riera-Lizarazu, Christopher C Mundt

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Article in TAG. Theoretical and applied genetics. Theoretische und angewandte Genetik, 2011. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.

0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed
2.5field-weighted citation impact, top 11% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

5 citing papers in PubMed, 11 citations in OpenAlex.

  1. Review
  2. Multi-location wheat stripe rust QTL analysis: genetic background and epistatic interactions.TAG. Theoretical and applied genetics. Theoretische und angewandte Genetik · 2015
    Article
  3. Article
  4. Mapping resistance genes for Oculimacula acuformis in Aegilops longissima.TAG. Theoretical and applied genetics. Theoretische und angewandte Genetik · 2014
    Article
  5. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

7 authors at 4 institutions in 3 countries.

Martin C QuinckeDepartment of Crop and Soil Science, Oregon State University, Corvallis, OR 97331, USA.
C James Peterson
Robert S Zemetra
Jennifer L Hansen
Jianli Chen
Oscar Riera-Lizarazu
Christopher C Mundt
Oregon State University · USUniversity of Idaho · USInternational Crops Research Institute for the Semi-Arid Tropics · INWashington State University · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Cephalosporium stripe, caused by Cephalosporium gramineum, can cause severe loss of wheat (Triticum aestivum L.) yield and grain quality and can be an important factor limiting adoption of conservation tillage practices. Selecting for resistance to Cephalosporium stripe is problematic; however, as optimum conditions for disease do not occur annually under natural conditions, inoculum levels can be spatially heterogeneous, and little is known about the inheritance of resistance. A population of 268 recombinant inbred lines (RILs) derived from a cross between two wheat cultivars was characterized using field screening and molecular markers to investigate the inheritance of resistance to Cephalosporium stripe. Whiteheads (sterile heads caused by pathogen infection) were measured on each RIL in three field environments under artificially inoculated conditions. A linkage map for this population was created based on 204 SSR and DArT markers. A total of 36 linkage groups were resolved, representing portions of all chromosomes except for chromosome 1D, which lacked a sufficient number of polymorphic markers. Quantitative trait locus (QTL) analysis identified seven regions associated with resistance to Cephalosporium stripe, with approximately equal additive effects. Four QTL derived from the more susceptible parent (Brundage) and three came from the more resistant parent (Coda), but the cumulative, additive effect of QTL from Coda was greater than that of Brundage. Additivity of QTL effects was confirmed through regression analysis and demonstrates the advantage of accumulating multiple QTL alleles to achieve high levels of resistance.

Indexed as

Immunity, InnateQuantitative Trait LociAcremoniumChromosome MappingChromosomes, PlantCrosses, GeneticDNA, PlantGenes, PlantGenetic MarkersPlant DiseasesPolymorphism, GeneticTriticumDNA, PlantGenetic Markers

Identifiers

PMID21258997
OpenAlexW1971807962

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.