Evidence map›Paper›PMID 21207010›Full record

ReviewDer Urologe. Ausg. A2011

[Drug therapy options for oligoasthenoteratozoospermia syndrome].

M Trottmann, F M Köhn, M Dickmann, C G Stief, A J Becker

Abstract readEnglish AbstractReview
PubMed Publisher
In one paragraph

Review in Der Urologe. Ausg. A, 2011. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 1 paper.

0numbers the graph read from it
0cells of the map it votes in
1citing papers in PubMed
–field-weighted citation impact, top 98% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

1 citing paper in PubMed, 2 citations in OpenAlex.

  1. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 2 institutions in 1 country.

M TrottmannUrologische Klinik und Poliklinik, Ludwig-Maximilians-Universität München, Campus Großhadern, Klinikum der Universität München, Marchioninistraße 15, 81377 München, Deutschland. matthias.trottmann@uro.med.uni-muenchen.de
F M Köhn
M Dickmann
C G Stief
A J Becker
LMU Klinikum · DEAndrologicum München

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Medical treatment of oligoasthenoteratozoospermia (OAT) syndrome includes many different treatment concepts. The targeted causes in the treatment of OAT syndrome are either hormonal, as for example hypothalamic, pituary, or, seldom, hyperprolactinemic disturbances, or acute or chronic urogenital infections, as well as ejaculatory disorders. Often no pathogenic reason for OAT syndrome can be found and targeted. Most treatment concepts in the past tried to improve sperm quality. Administration of antiestrogens, antioxidants, hormones, and micronutrients has been tried and used to improve sperm quality. The present article provides an overview of current medical treatment options for OAT syndrome, including former unsuccessful treatment concepts. The article furthermore sketches out potential treatment options, which might be available in the future.

Indexed as

AntioxidantsAsthenozoospermiaHormone Replacement TherapyHumansHyperprolactinemiaInfertility, MaleMaleSyndromeTesticular DiseasesTreatment OutcomeUrinary Tract InfectionsAntioxidants

Identifiers

PMID21207010
OpenAlexW177208790

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.